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Huberman Lab - Essentials: Using Salt to Optimize Mental & Physical Performance
Episode Date: March 26, 2026In this Huberman Lab Essentials episode, I explain how salt (sodium) affects mental and physical performance, as well as cellular health. I describe how the brain monitors sodium levels to regulate th...irst and fluid balance, and why salt needs can vary depending on activity level, stress, blood pressure, and diet. I also explain how to determine the right sodium intake for your individual needs and discuss why some people may benefit from increasing salt and other electrolytes. Read the episode show notes at hubermanlab.com. Thank you to our sponsors AG1: https://drinkag1.com/huberman Function: https://functionhealth.com/huberman LMNT: https://drinklmnt.com/huberman Timestamps (00:00:00) Salt (00:00:37) Brain & Monitoring Salt (00:02:33) Thirst, Osmotic Thirst & Salt (00:05:35) Hypovolemic Thirst & Blood Pressure (00:06:59) Sponsor: Function (00:08:39) Fluid Balance, Kidney & Urine Regulation (00:11:53) How Much Salt Do You Need?, Blood Pressure, Dizziness & Postural Syndromes (00:17:29) Replenish Salt for Performance, Tool: Galpin Equation & Exercise (00:19:15) Sponsor: LMNT (00:20:46) Stress & Craving Salt (00:22:29) Electrolytes: Magnesium & Potassium; Low Carbohydrate Diet (00:25:19) Salt & Sweet Taste, Sugar Cravings, Processed Foods (00:29:37) Finding Your Ideal Salt Intake, Tool: Unprocessed Food Diet (00:31:25) Sponsor: AG1 (00:32:50) Neurons, Salt & Action Potentials; Ingesting Too Much Water (00:34:51) Recap & Key Takeaways Disclaimer & Disclosures Learn more about your ad choices. Visit megaphone.fm/adchoices
Transcript
Discussion (0)
Welcome to Huberman Lab Essentials, where we revisit past episodes for the most potent and actionable science-based tools for mental health, physical health, and performance.
I'm Andrew Huberman, and I'm a professor of neurobiology and ophthalmology at Stanford School of Medicine.
Today we are going to discuss salt, also referred to as sodium.
Salt has many, many important functions in the brain and body.
For instance, it regulates fluid balance, how much fluid you desire and how much fluid you desire and how
how much fluid you excrete.
Salt also regulates your appetite for other nutrients,
things like sugar, things like carbohydrates.
We all harbor small sets of neurons.
We call these sets of neurons nuclei,
meaning little clusters of neurons,
that sense the levels of salt in our brain and body.
There are a couple brain regions that do this,
and these brain regions are very, very special,
special because they lack biological fences
around them that other brain areas have.
have and those fences, or I should say that fence
goes by a particular name and that name is the blood brain barrier
or BBB.
Most substances that are circulating around in your body
do not have access to the brain,
in particular large molecules,
can't just pass into the brain.
The brain is a privileged organ in this sense.
However, there are a couple of regions in the brain
that have a fence around them,
but that fence is weaker.
And it turns out that the areas of the brain,
that monitor salt balance and other features
of what's happening in the body at the level
of what we call osmalarity,
at the concentration of salt,
reside in these little sets of neurons
that sit just on the other side of these weak fences.
And the most important and famous of these
for its sake of today's conversation is one called OVLT.
OVLT stands for the organum vascularum of the lateral terminus.
The neurons in that,
that region are able to pay attention
to what's passing through in the bloodstream
and can detect for instance if the levels of sodium
in the bloodstream are too low,
if the level of blood pressure in the body is too low or too high,
and then the OVLT can send signals to other brain areas,
and then those other brain areas can do things like release hormones
that can go and act on tissues in what we call the periphery
in the body, for instance, have the kidneys secrete more urine
to get rid of salt that's excessive salt in the body.
So let's talk about the function of the OVLT
and flesh out some of the other aspects of its circuitry,
of its communication with other brain areas
and with the body in the context of something
that we are all familiar with, which is thirst.
Have you ever wondered just why you get thirsty?
Well, it's because neurons in your OVLT
are detecting changes in your bloodstream,
which detect global changes within your body.
And in response to that, your OVLT sets
off certain events within your brain and body
that make you either want to drink more fluid
or to stop drinking fluid.
There are two main kinds of thirst.
The first one is called osmotic thirst
and the second is called hypovolemic thirst.
Osmotic thirst has to do with the concentration
of salt in your bloodstream.
So let's say you ingest something very, very salty.
Let's say you ingest a big bag of,
I confess I don't eat these very often
but I really like those kettle potato chips.
And I don't have too much shame about that
because I think I have a pretty healthy relationship to food
and I enjoy them and I understand that it will drive salt levels
up in my bloodstream and that will cause me to be thirsty.
But why?
Why?
Because neurons in the OVLT come in two main varieties.
One variety senses the osmilarity of the blood
and when the osmolarity meaning the salt concentration
in the blood is high, it activates these specific neurons,
in the OVLT, and by activates,
I mean it causes them to send electrical potentials,
literally send electrical signals to other brain areas.
And those other brain areas inspire a number
of different downstream events.
The consequence of that communication
is that a particular hormone is eventually released
from the posterior pituitary.
So from the pituitary, there's a hormonal signal
that's released called vasopressin.
Vesopressin also goes by the name
anti-diuretic hormone and antidiarrotic hormone has the capacity to either restrict the amount
of urine that we secrete or when that system is turned off to increase the amount of urine
that we secrete. So there's a complicated set of cascades that's evoked by having high salt
concentration in the blood. There's also a complicated set of cascades that are evoked by having low
concentrations of sodium in the blood. But the pathway is nonetheless the same.
Its OVLT is detecting those osmolarity changes, communicating to the superoptic nucleus.
Superoptic nucleus is either causing the release of or is releasing vasopressin, anti-diarrotic hormone,
or that system is shut off so that the anti-diuretic hormone is not secreted,
which would allow urine to flow more freely, right? Antidiarretic means anti-relierecteretic means anti-release
of urine and by shutting that off, you're going to cause the release of urine. You're sort of
allowing a system to flow, so to speak. The second category of thirst is hypovolemic thirst.
Hypovolemic thirst occurs when there's a drop in blood pressure. Okay, so the OVLT, as I mentioned
before, can sense osmilarity based on the fact that it has these neurons that can detect
how much salt is in the bloodstream. But the OVLT also harbors neurons that,
that are of the baro receptor mechanoreceptor category.
Now, more on barro receptors and mechanoreceptors later,
but barro receptors are essentially a receptor,
meaning a protein that's in a cell,
that responds to changes in blood pressure.
So there are a number of things that can cause decreases
in blood pressure.
Some of those include, for instance,
if you lose a lot of blood, right,
if you're bleeding quite a lot,
or in some cases,
if you vomit quite a lot,
or if you have extensive diarrhea,
or any combination of those,
both types of thirst, osmotic thirst and hypovolemic thirst,
are not just about seeking water,
but they also are about seeking salt.
In very general terms, salt, aka sodium,
can help retain water,
but sodium and water work together
in order to generate what we call thirst.
Sodium water work together
in order to either retain water
or inspire us to let go of water to urinate.
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early access to function. So before we can dive into the specifics around salt and how to use
salt for performance and various recommendations and things to avoid, we need to drill a little bit
deeper into this fluid balance mechanism in the body. And for that reason, we have to pay at least a little
bit of attention to the kidney. The kidney is an incredible organ. And one of the reasons the kidney
is so amazing is that it's responsible for both retaining holding onto or allowing the release
of various substances from the body. Basically, blood enters the kidney and it goes through a series
of tubes, which are arranged into loops. If you want to look more into this, there's the beautiful
loop of Henley and other aspects of the kidney design that allow,
certain substances to be retained and other substances to be released, depending on how concentrated
those substances are in the blood. The kidney responds to a number of hormonal signals, including
vasopressin, in order to, for instance, antidiarratic hormone, in order to hold on to more fluid,
if that's what your brain and body need. And it responds to other hormonal signals as well. So it's a
pretty complex organ. So the way the kidney is designed is that about 90% of, you know,
of the stuff that's absorbed from the blood
is going to be absorbed early in this series of tubes.
So just to give a really simple example,
let's say that you are very low on fluid.
You haven't had much to drink in a while,
maybe you're walking around on a hot day.
Chances are that the neurons in your OVLT
will sense the increase in osmilarity, right?
The concentration of salt is going to be increased
relative to the fluid volume that's circulating.
This, of course,
that you haven't excreted a lot of sodium
for one reason or another.
But that increase in osmalarity is detected by the OVLT.
The OVLT is going to signal a bunch of different cascades
through the super optic nucleus, et cetera.
And then vasopressin is going to be released
into the bloodstream.
And vasopressin, again, also called antidioretic hormone,
is going to act on the kidney
and change the kidneys function in a couple of different ways,
some mechanical, some chemical, okay,
in order to make sure that your kidney does not release much water.
Doesn't make you want to urinate.
And in fact, even if you would try to urinate,
your body's gonna tend to hold on to its fluid stores.
Okay, so very simple, straightforward example.
We can also give the other example whereby,
if you're ingesting a lot, a lot, a lot of water,
and it's not a particularly hot day
and you're not sweating very much.
Let's assume your salt intake is constant
or is low for whatever reason.
Well, then the osmilarity, the salt concentration,
in your blood is going to be lower.
Your OVLT will detect that because of these osmosensing neurons
in your OVLT.
Your OVLT will fail to signal to the superoptic nucleus
and there will not be the release of vasopressin
antidearetic hormone and you can excrete all the water
that your body wants to excrete.
Meaning you'll be able to urinate.
There's no holding on to water at the level of the kidney.
Okay.
So how much salt do we need?
And what can we trust in terms of trying
to guide our ingestion of salt?
First of all, I wanna be very, very clear
that there are a number of people out there
that have prehypertension or hypertension.
You need to know if you have prehypertension or hypertension.
You need to know if you have normal tension,
meaning normal blood pressure.
Everyone should know their blood pressure.
It's an absolutely crucial measurement
that has a lot of impact
on your immediate and long-term health outcomes.
It informs a lot about
what you should do.
Should you be doing more cardiovascular exercise?
Should you be ingesting more or less salt?
And without knowing what your blood pressure is,
I can't give a one size fits all recommendation.
And indeed, I'm not gonna give medical recommendations.
I'm simply gonna spell out what I know about the research,
which hopefully will point you in the direction
of figuring out what's right for you
in terms of salt and indeed fluid intake.
There is a school of thought
that everybody is consuming too much salt.
And I do want to highlight the fact
that there are dozens, if not hundreds of quality papers
that point to the fact that a quote unquote high salt diet
can be bad for various organs and tissues in the body,
including the brain.
It just so happens that because fluid balance,
both inside and outside of cells is crucial,
not just for your heart and for your lungs
and for your liver and for all the organs of your body,
but also for your brain that if the salt constant,
salt concentration inside of cells in your brain becomes too high,
neurons suffer.
They will draw fluid into those cells
because water tends to follow salt, as I mentioned before,
and those cells can swell.
You can literally get swelling of brain tissue.
Conversely, if salt levels are too low inside of cells
in any tissue of the body, but in the brain included,
then the cells of the body,
body and brain can shrink because water is pulled
into the extra cellar space away from cells.
And indeed, under those conditions, brain function can suffer.
And indeed, the overall health of the brain can suffer.
At fairly low levels of sodium,
meaning at about two grams per day,
you run fewer health risks,
but the number of risk continues to decline
as you move towards four and five grams per day.
And then as you increase your salt intake
further than the risk dramatically increases.
Most people are probably consuming more than that
because of the fact that they are ingesting processed foods
and processed foods tend to have more salt in them
than non-processed foods.
But if we are to take this number of 2.3 grams,
that's the recommended cutoff for ingestion of sodium,
that indeed is associated with low incidence
of hazardous outcomes, cardiovascular vent stroke, et cetera,
So again, I want to be very, very clear that you need to know your blood pressure.
If you have high blood pressure or your pre-hypertensive, you should be especially cautious
about doing anything that increases your blood pressure.
And as always, you want to, of course, talk to your doctor about doing anything that could
adjust your health in any direction.
But there are a number of people out there that have low blood pressure, right?
People that get dizzy when they stand up.
People that are feeling chronically fatigued.
And in some cases, not all.
Those groups can actually benefit from increasing their sodium intake.
take. Why? Well, because of the osmilarity of blood that we talked about before, where if you have
a certain concentration of sodium, meaning sufficient sodium in your bloodstream, that will tend to
draw water into the bloodstream. And essentially, the pipes that are your capillaries, arteries,
and veins will be full. The blood pressure will get up to your head, whereas some people, their
blood pressure is low because the osmolarity of their blood is low. And that can have a number of
downstream consequences. I should also mention it can be
the consequence itself of challenges or even deficits in kidney function.
But all of these organs are working together.
So the encouragement here is not necessarily to ingest more sodium.
It's to know your blood pressure and to address whether or not an increase in sodium intake
would actually benefit your blood pressure in a way that could relieve some of the dizziness
and other symptoms of things like orthostatic disorders.
Let's look at what the current recommendations are for people that suffer
from orthostatic disorders like orthostatic hypo,
meaning too low tension, orthostatic hypotension,
postural tachycardia syndrome,
sometimes referred to as pots, P-O-T-S,
or idiopathic orthostatic tachycardia and syncope.
Those groups are often told to increase their salt intake
in order to combat their symptoms.
The American Society of Hypertension recommends anywhere
from 6,000 to 10,000.
These are very high levels.
So this is six grams to 10 grams of salt per day.
keeping in mind again that salt is not the same as sodium.
So that equates to about 2,400 to 4,000 milligrams
of sodium per day.
I point out this paper and I point out
these higher salt recommendations to emphasize again
that context is vital, right?
That people with high blood pressure
are going to need certain amounts of salt intake.
People with lower blood pressure
are going to need higher amounts of salt.
And for most people out there,
you're going to need to evaluate how much salt intake
is going to allow your brain and body to function optimally.
So if you're exercising a lot,
if you're a particular cold, dry environment,
or a particular hot environment,
you ought to be ingesting sufficient amounts of salt and fluid.
A rule of thumb for exercise-based replenishment of fluid
comes from what I, some episodes back,
referred to as the Galpin equation.
The Galpin equation, I named it after Andy Galpin,
and I think that is the,
appropriate attribution there.
Andy Galpin is an exercise physiologist.
So the Galpin equation is based on the fact
that we lose about one to five pounds of water per hour,
which can definitely impact our mental capacity
and our physical performance.
And the reason that loss of water from our system
impacts mental capacity and physical performance
has a lot to do with literally the changes
in the volume of those cells, the size of those cells,
based on how much sodium is contained in or outside those cells,
and the formula for hydration,
the so-called Galpin equation,
is your body weight in pounds divided by 30
equals the ounces of fluid
you should drink every 15 minutes.
Now, the Galpin equation is mainly designed for exercise,
but I think is actually a very good rule of thumb
for any time that you need to engage mental capacity,
not just physical performance.
The idea is to make sure that you're entering the activity,
cognitive or physical, sufficiently hydrated,
and that throughout that activity you're hydrating regularly.
And it points to the fact that most people are probably underhydrating,
but not just underhydrating from the perspective of not ingesting enough water,
that they're probably not getting enough electrolytes as well,
sodium, potassium and magnesium.
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So we've all heard about how excess salt,
it's bad for blood pressure, damage the heart, the brain, et cetera.
I do want to give some voice to situations where too little salt can actually cause problems.
And this has everything to do with the nervous system.
So without getting into excessive amounts of detail, the kidneys, as we talked about before,
are going to regulate salt and fluid balance.
The adrenal glands, which right atop the kidneys, are going to make glucocorticoids like aldosterone.
and those are going to directly impact things like fluid balance.
And in part they do that by regulating how much craving for and tolerance of salty solutions we have.
The whole basis for a relationship between the adrenal system, these glucocorticoids, things like aldostron, and the craving for sodium, is that the stress system is a generic system designed to deal with various challenges to the organism, to use.
or to me or to an animal.
And those challenges can arrive in many different forms.
It can be an infection, it can be famine,
it can be lack of water, and so on.
But in general, the stress response is one of elevated heart rate,
elevated blood pressure,
and an ability to maintain movement and resistance to that challenge.
It's clear from a number of studies that if sodium levels are too low,
that our ability to meet stress challenges is impaired.
There are conditions such as when we are under stress challenge, when there is a natural craving for more sodium, and that natural craving for more sodium is hardwired into us as a way to meet that challenge.
Now, we can't have a discussion about sodium without having a discussion about the other electrolytes, magnesium and potassium.
I want to emphasize that many people are probably getting enough magnesium in their diet that they don't need to supplement magnesium.
Some people, however, opt to supplement magnesium
in ways that can support them.
And there are many different forms of magnesium.
And just in very brief passing,
I'll just say that there is some evidence
that you can reduce muscle soreness from exercise
by ingestion of magnesium malate, M-A-T-E.
I've talked before about magnesium threonate,
T-H-R-E-E-N-O-A-T-E,
magnesium threonate for sake of promoting the transition
into sleep and for depth of sleep.
And then there are other forms of magnesium,
magnesium bisglycinate, which it seems at least on par
with magnesium three and eight in terms of promoting
transition into in depth of sleep and so on.
There are other forms of magnesium, magnesium citrate,
which has other functions.
Actually, magnesium citrate is a fairly effective laxative,
not known to promote sleep and things of that sort.
So a lot of different forms.
of magnesium and there's still other forms out there.
Many people are not getting enough magnesium, many people are.
Okay, so that's magnesium.
Anytime we're talking about sodium balance,
we have to take into consideration potassium
because the way that the kidney works
and the way that sodium balance is regulated
both in the body and the brain
is that sodium and potassium are working in close concert
with one another.
There are a lot of different recommendations
about ratios out there and they range widely
from two to one ratio
of potassium to sodium.
I've heard it in the other direction too.
I've heard a two to one sodium to potassium.
The recommendations vary.
Now for people that are following low carbohydrate diets,
one of the most immediate effects of a low carbohydrate diet
is that you're gonna excrete more water.
And so under those conditions,
you're also going to lose, not just water,
but you'll probably also lose sodium and potassium.
And so some people, many people in fact,
find that when they are on a lower or low carbohydrate diet,
then they need to make sure that they're getting enough sodium and enough potassium.
And of course, some people who are on low carbohydrate diets do ingest vegetables,
you know, or other forms of food that carry along with them, potassium.
So it's quite variable from person to person.
I mean, you can imagine if carbohydrate holds water,
water and salt balance and potassium go hand in hand and hand,
that if you're on a low carbohydrate diet,
that you might need to adjust your salt intake and potassium.
And conversely, that if you're on a carbohydrate-rich diet
or a moderate carbohydrate diet,
then you may need to ingest less sodium and less potassium.
So up until now, we've been talking about salt as a substance
and a way to regulate fluid balance and blood volume and so on.
We haven't talked a lot about salt as a taste
or the taste of things that are salty.
And yet we know that we have salt receptors,
meaning neurons, that fire action potentials,
when salty substances are detected,
much in the same way that we have sweet detectors
and bitter detectors,
and we have detectors of umami, the savory flavor on our tongue.
Well, we also have salt sensors at various locations
throughout our digestive tract,
although that the sensation and the taste of salt
actually exerts a very robust effect
on certain areas of the brain
that can either make us crave more
or sate, meaning fulfill our desire for salt.
And you can imagine why this would be
important, your brain actually has to register whether or not you're bringing in salt in
order to know whether or not you are going to crave salt more or not. And beautiful work that's
been done by the Zucker Lab, ZUKER, ZUKER, Zucker Lab at Columbia University, as well as many other labs
have used imaging techniques and other techniques such as molecular biology to define these
so-called parallel pathways. Parallel meaning pathways that represent sweet or the presence
of sweet taste in the mouth and gut. Parallel pathways, meaning,
neural circuits that represent the presence of salty tastes
in the mouth and gut and so on.
And that those go into the brain, move up through brain stem centers
and up to the neocortex, indeed where our seed
of our conscious perception is,
to give us a sense and a perception of the components
of the foods that we happen to be ingesting.
The pathways, the parallel pathways for salty
and the parallel pathways for sweet and bitter
and so on can actually interact.
And this has important
relevance in the context of food choices and sugar craving.
One of the things that's commonplace nowadays
is in many processed foods, there is a business, literally,
a business of putting so-called hidden sugars.
And these hidden sugars are not always
in the form of caloric sugars.
They're sometimes in the form of artificial sweeteners
into various foods.
And you might say, well, why would they put more sugar
into a food and then disguise the sugary taste,
given that sweet taste often compel people
to eat more of these things.
things. Well, it's a way actually of bypassing some of the homeostatic mechanisms for sweet.
You know, even though we might think that the more sweet stuff we eat, the more sweet stuff
we crave, in general, people have a threshold whereby they say, okay, I've had enough
sugary stuff. So these sensory systems interact in this way. By putting sugars into foods
and hiding the sugary taste of those foods, those foods, even if they contain artificial
sweeteners, that will then signal to the brain to,
to release more dopamine and make you crave more of that food.
Whereas had you been able to perceive the true sweetness
of that food, you might have consumed less.
And indeed, that's what happens.
So these hidden sugars are kind of diabolical.
Why am I talking about all of this
in the context of an episode on salt?
Well, as many of you probably noticed,
a lot of foods out there contain a salty, sweet combination.
And it is that combination of salty and sweet,
which can actually lead you to consume more
of the salty sweet food than you would have
if it had just been sweet or it had just been salty.
And that's because both sweet taste and salty taste
have a homeostatic balance.
So if you ingest something that's very, very salty,
pretty soon your appetite for salty foods will be reduced.
But if you mask some of that with sweet,
well, because of the interactions of these parallel pathways,
you somewhat shut down your perception
of how much salt you're ingesting.
or conversely, by ingesting some salt with sweet foods,
you mask some of the sweetness of the sweet foods
that you're tasting and you will continue
to indulge in those foods.
So salty sweet interactions can be very diabolical.
They can also be very tasty,
but they can be very diabolical in terms of inspiring you
to eat more of a particular food
than you would otherwise if you were just following
your homeostatic salt or your homeostatic sugar balance systems.
So your brain has a way of representing
the pure form of taste.
salty, sweet, bitter, et cetera,
and has a way of representing their combinations.
And food manufacturers have exploited this to large degree.
I mention all of this because if you're somebody
who's looking to explore either increasing
or decreasing your sodium intake for health benefits,
for performance benefits, in many ways,
it is useful to do that in the context
of a fairly pure, meaning unprocessed food intake background,
whether or not that's keto, carnivore,
omnivore, intermittent fasting or what have you.
It doesn't really matter.
But the closer that foods are to their basic form and taste,
meaning not large combinations of large amounts
of ingredients and certainly avoiding highly processed foods,
the more quickly you're going to be able to hone in
on your specific salt appetite and salt needs,
which as I've pointed out numerous times throughout this episode
are going to vary from person to person,
depending on nutrition, depending on activity,
depending on hormone status.
So if you want to home in on the appropriate amount of sodium for you,
yes, blood pressure is going to be an important metric
to pay attention to as you go along.
But in determining whether or not increasing your salt intake
might be beneficial for, for instance,
for reducing anxiety a bit or for increasing blood pressure
to offset some of these postural syndromes
where you get dizzy, et cetera,
for improving sports performance or cognitive performance.
And indeed, many people find,
and it's reviewed a bit, and some of the data
are reviewed in the book, the salt fix,
that when people increase their salt intake
in a backdrop of relatively unprocessed foods,
that sugar cravings can indeed be vastly reduced.
And that makes sense given the way
that these neural pathways for salty and sweet interact.
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Now, thus far, I've already covered quite a lot of material,
but I would be completely remiss if I didn't emphasize
the crucial role that sodium plays in the way that neurons function.
In fact, sodium is one of the key elements
that allows neurons to function at all.
And that's by way of engaging what we call the admonies,
action potential.
The action potential is the fundamental way
in which neurons communicate with one another.
The point I'd like to make,
at least as it relates to this episode on salt,
is that having sufficient levels of salt in your system
allows your brain to function,
allows your nervous system to function at all.
Again, this is the most basic aspect
of nervous system function.
And there are cases where this whole system gets disrupted.
And that brings us to the topic of sodium
water balance.
As many of you have probably heard,
but hopefully if you haven't,
you'll take this message seriously.
If you drink too much water,
especially in a short amount of time,
you can actually kill yourself, right?
And we certainly don't want that to happen.
If you ingest a lot of water in a very short period of time,
something called hypernatremia,
you will excrete a lot of sodium very quickly
and your ability to regulate kidney function
will be disrupted,
But in addition to that, your brain can actually stop functioning.
And I've talked about this a bit in the episode on endurance,
but there are instances in which competitive athletes
have come into the stadium to finish a final lap
of a long endurance race and are completely disoriented
and actually can't find their way to the finish line.
You know, it might sound like kind of a silly, crazy example,
but there are examples of people having severe mental issues
and physical issues post exercise
when that exercise involved a ton of sweat,
or hot environments or insufficient ingestion of fluids
and electrolytes because included in the electrolyte formula,
of course, is sodium.
And as you just learned, sodium is absolutely crucial
for neurons to function.
So to briefly recap some of what I've talked about today,
we talked about how the brain monitors the amount of salt
in your brain and body and how that relates to thirst
and the drive to consume more fluid and or salty fluids.
We also talked a little bit about the hormones
that come from the brain and operated
the level of the kidney in order to either retain
or allow water to leave your system.
Talked a little bit about the function of the kidney itself,
a beautiful organ.
We talked about the relationship between salt intake
and various health parameters
and how a particular range of salt intake
might be optimal depending on the context
in which that range is being consumed.
Meaning depending on whether or not your hyperticarious,
hypertensive, prehypertensive, or normal tension.
We talked about fluid intake and electrolyte intake,
so sodium, pentasm, and magnesium
in the context of athletic or sports performance,
but also in terms of maintaining cognitive function.
Talk about the Galpin equation,
which you could easily adapt to your body weight
and to your circumstances.
Of course, adjusting the amount of fluid
and electrolyte intake upwards
if you're exercising or working in very hot environments,
Downwards, maybe if you're in less hot environments
where you're sweating less and so on.
We also talked about the relationship
between the stress system and the salt craving system
and why those two systems interact
and why for some people who may suffer a bit
from anxiety or under conditions of stress,
increasing salt intake provided it's done
through healthy means might actually be beneficial.
We also talked about conditions
in which increasing salt intake might be beneficial
for offsetting low blood pressure
and some of these postural syndroms
that can lead people to dizziness and so forth.
These are things that have to be explored
on an individual basis and of course,
have to be explored with the support of your doctor.
We also talked about the perception of salt,
meaning the perception of salty tastes
and how the perception of salty taste
and the perception of other tastes like sweet
can interact with one another
to drive things like increased sugar intake
when you're not even aware of it.
And indeed how the combination
salty and sweet taste can bias you towards craving more,
for instance, processed foods
and why that might be a good thing to avoid.
And of course, we talked about salt
and its critical role in the action potential,
the fundamental way in which the nervous system functions at all.
So my hope for you in listening to this episode
is that you consider a question.
And that question is, what salt intake is best for you?
And that you place that question
in the context of your fluid intake.
And crucially, that you,
you place that in the context of the electrolytes more generally,
meaning sodium, potassium, and magnesium.
And I hope I've been able to illuminate
some of the beautiful ways in which the brain
and the bodily organs interact
in order to help us regulate this thing
that we call sodium balance.
And the fact that we have neurons in our brain
that are both tuned to the levels of salt in our body
and position in a location in the brain
that allows them to detect the levels of salt in our body
and to drive the intake of more or more
or less salt and more or less fluid and other electrolytes,
really just points to the beauty of the system
that we've all evolved that allows us to interact
with our environment and make adjustments
according to the context of our daily and ongoing life.
And last but certainly not least,
thank you for your interesting science.
