Huberman Lab - Essentials: Science of Building Strong Social Bonds with Family, Friends & Romantic Partners
Episode Date: November 20, 2025In this Huberman Lab Essentials episode, I discuss the science of social connection and how we form meaningful bonds with others. I explore the neural basis for "social homeostasis"—our drive for a... certain amount of social interaction—which explains why we feel lonely, seek connection and how we navigate social hierarchies. I also explain how the brain and neurochemicals, such as oxytocin and dopamine, shape our relationships from infancy through adulthood and underlie traits like introversion and extroversion. The episode also offers practical insights into forming deeper connections and how shared experiences with others enhance social bonding. Read the episode show notes at hubermanlab.com. Thank you to our sponsors AGZ by AG1: https://drinkagz.com/huberman LMNT: https://drinklmnt.com/huberman David: https://davidprotein.com/huberman Timestamps 0:00 Social Connection 1:10 Social Bonds, Social Isolation & Stress Hormones 3:09 Sponsor: LMNT 4:42 Brain & Social Homeostasis; Social Hierarchies & Flexibility 9:14 Dopamine & Pro-Social Behaviors; Chronic Social Isolation & Introversion 11:04 Introverts vs Extroverts, Dopamine & Social Homeostasis; Context 13:08 Loneliness, Dorsal Raphe Nucleus & Social Hunger 14:18 Key Takeaway: Introvert vs Extrovert & Dopamine 15:23 Social Bonds & Physiological Synchrony, Tool: Shared Experiences 18:19 Sponsor: AGZ by AG1 19:48 Right- vs Left-Brained Attachment, Parent & Child, Unconscious Mind 24:30 Friends & Romantic Partners, Emotional & Cognitive Empathy 27:52 Sponsor: David 29:09 Oxytocin & Social Connection 31:39 Tool: Emotional & Cognitive Empathy 32:54 Introverts, Extroverts & Social Interaction 33:48 Break-Ups; Key Takeaways Disclaimer & Disclosures Learn more about your ad choices. Visit megaphone.fm/adchoices
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Welcome to Huberman Lab Essentials,
where we revisit past episodes
for the most potent and actionable science-based tools
for mental health, physical health, and performance.
I'm Andrew Huberman, and I'm a professor of neurobiology
and ophthalmology at Stanford School of Medicine.
Today's episode is about the biology, psychology,
and practices of social bonding.
From the day we are born until the day we die,
the quality of our social bonds dictates much of our quality of life.
our quality of life.
It should therefore be no surprise
that our brain and indeed much of our entire nervous system
is wired for social bonds.
Today we are going to talk about those brain
and nervous system circuitries.
We're also going to talk about the neurochemicals
and hormones that underlie their function.
And we are going to touch on a number of important
and actionable tools that you can apply in everyday life
and I'm confident that you will come away
from today's episode with tremendous knowledge
about how you function.
For instance, if you're an introvert or an extrovert,
why is that?
Turns out there may be a neurochemical basis for that.
Believe it or not, there's biology around that now
and it's excellent peer reviewed work.
Now, an important feature of biology generally,
but in particular as it relates to social bonding,
is that the neural circuits,
meaning the brain areas and neurons and the hormones,
things like oxytocin, which we'll talk about today,
and the other chemicals in the brain and body
that are responsible for the process we call social,
are not unique to particular social bonds.
They are generic.
What I mean by that is that the same brain circuits
that are responsible for establishing a bond
between parent and child are actually repurposed
in romantic relationships.
Before we talk about social bonding,
I wanna talk about it to mirror image,
which is lack of social bonding or social isolation.
Many people like time alone.
But when we talk about social isolation,
what we're referring to is,
when animals or humans are restricted
from having the social contacts that they would prefer to have.
And to just briefly touch on the major takeaways
from this literature, which spans back 100 years or more,
being socially isolated is stressful.
And one of the hallmark features of social isolation
is chronically elevated stress hormones,
like adrenaline, also called epinephrine,
like cortisol, a stress hormone that at healthy levels
is good for combating inflammation,
helps us have energy early in the day,
focus throughout the day.
But if cortisol is elevated for too long,
which is the consequence of social isolation,
the immune system suffers and other chemicals
start to be released in the brain and body
that are designed to motivate the organism,
animal or human, to seek out social bonds.
So if you're somebody who's socially isolated
and is craving social contact,
that is a healthy craving.
And as we'll learn next,
The healthy craving for social contact
has a very specific brain circuit,
has a very specific neurochemical signature associated with it,
and has some remarkable features
that you can leverage in social contacts of all kinds.
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Much like hunger, much like temperature, much like thirst, we have brain circuits that are devoted
to what's called a social homeostasis.
Homeostasis is the characteristic
of various biological circuits
and even individual cells to try and maintain a certain level.
It's most easily thought of in the context of hunger.
If you don't eat for a while,
your drive to pursue food and think about food
and make food and spend money on food
and indeed to enjoy food goes up.
Whereas when you're well fed,
you don't tend to seek out food
with as much vigor or as much intensity.
So that's a simple way of thinking about homeostasis.
Every homeostatic circuit has three components,
or at least three.
One is a detector, meaning the organism
or the thermostat on your wall
has to have some way of detecting
what's going on in the environment, all right,
in the context of social bonding,
whether or not you are interacting with others
and whether or not those interactions are going well.
So that has to be detected, that's the first thing.
Then there has to be a control center.
That's the second thing.
And the control center is the one
that makes the adjustments to,
to, in the case of social bonding,
to your behavior and to your psychology.
Now the third component of this homeostatic circuit
is the effector.
The effector is actually what drives the behavioral response.
It's what leads you to pick up your social media
and start scrolling.
It's what leads you to text a friend.
So again, those three components are a detector,
a control center, and an effector.
And as you'll soon learn, the neural circuit
that controls this social homeostasis,
actually has a fourth component,
And that fourth component is one that places subjective understanding as to why you are doing
what you are doing and establishes your place in a hierarchy.
When we talk about social hierarchies in the context of human interactions, social hierarchies
are very plastic, meaning in one setting, one person can be the leader.
In another setting, the other person can be the leader.
Hierarchies are very dynamic.
And as a consequence, social bonding has to be very plastic and very fluid.
fluid so that you move from one environment to the next,
even with the same people,
you have to be able to make those adjustments.
And in the case of the social homeostasis circuit,
those adjustments are made by a particular brain structure.
I've talked about on this podcast before.
It's called the prefrontal cortex.
It is the seat of our higher consciousness, if you will.
It's what allows us to place subjective labels on things.
So we are not strictly input output.
We are not robotic.
The detector that underlies social homeostasis involves,
mainly two structures.
One is called the ACC, the anterior cingulate cortex,
and the other is the BLA, basilateral amygdala.
And when you hear the word amygdala,
you're probably thinking fear.
But today, as you'll see,
the amygdala actually has many different sub-compartments
and components.
And there's a reason why the basalateral amygdala,
which is associated with certain aspects
of aversive behaviors,
meaning moving away from certain types of things or interactions.
There's a reason why the BLA is such an integral part
of the detector system.
And that's because,
just as it's important to form healthy social bonds,
it's vitally important to try and avoid unhealthy social bonds.
So we've got the ACC and the BLA.
These are areas that are mainly involved in moving away from things,
although also toward them.
That's the detector.
Then we've got the control center, which is in the hypothalamus.
And then there's a very special and important area
associated with social bonding that I want everyone to learn,
which is the dorsal rafae nucleus or dRN,
dorsal raffae nucleus.
The dorsal raffae nucleus is a small collection of neurons
in the midbrain, so it's deep in the brain.
And most of the time when you hear about raffae,
are a P-H-E, by the way, raphae nucleus,
you're talking about serotonin.
Serotonin is a neuromodulator that is often associated
with feelings of satiety after eating,
basically satisfaction with things that you already have.
However, within this dorsal raffa nucleus,
there is a small set of,
set of neurons that release dopamine.
Dopamine is a neuromodulator most often associated
with movement, craving, motivation, and desire.
This unique population of dopamine neurons in the RAFA
is truly unique in that it's responsible
for mediating what I've been calling social homeostasis.
It is the effector or the response
that mediates social homeostasis.
In most popular conversations about dopamine
and even in scientific circles,
when you hear dopamine release,
you think about relostasis.
ward or feeling good.
However, dopamine is not associated with feeling good.
It is actually the neurochemical that's responsible
for movement toward things that feel good.
So to zoom out and conceptualize what we have here,
we have a brain area that is a detector
that either will move us toward or away
from certain types of experiences or sensations.
We have a control center that is going to release
certain hormones and neuropeptides
into our brain and blood,
depending on the sort of
sorts of interactions that we happen to be having.
And we have this response system,
which is the dorsal raffine nucleus that contains dopamine neurons.
If you're somebody who is accustomed to a lot of social interaction
and suddenly I take away that social interaction,
you would feel kind of let down.
You would crave a replacement social interaction.
Okay, this is called a pro-social craving.
And indeed, this is what you see in animals and humans.
If you, what's called,
acutely isolate them, which is just a fancy scientific word
of saying deprive them of social interactions
in a short-term basis, they start engaging
in pro-social behaviors.
So the takeaway is that when we lack social interaction
that we expect, we become prosocial.
However, if we are chronically socially isolated,
meaning we don't have interactions
with people for a long time,
we become actually more introverted.
It's well established now that in humans
and in animals, if you don't give them
enough social interaction, they actually become antisocial.
The social homeostasis circuit works in a way
such that when we don't have social interactions
for a very long time, we start to lose our craving
for social interactions.
Let's look at the social homeostasis circuit
through the lens of what's commonly called
introversion and extroversion.
Now, typically when we hear about introverts,
we think about the quiet person at the party
or the person that doesn't want to go out at all.
And we think about an extrovert
as somebody who's really social,
the so-called social butterfly,
who enjoys social interactions,
it's really chatty,
it's kind of life of the party type person.
But actually in the psychology literature,
that's not really the way it holds up.
If we look at introversion and extroversion
through this lens of the social homeostatic set point
and we think about dopamine as this molecule
that drives motivation to seek out social interactions,
what we can reasonably assume
is that introverts are people
that when they engage in certain forms of,
social interaction, either the amount of dopamine
that's released is greater than it is in an extrovert.
That's right, I said greater than it is in an extrovert.
And so they actually feel quite motivated,
but also satisfied by very brief
or we could say sort of sparse social interactions.
They don't need a lot of social engagement
to feel sated.
Whereas the extrovert, we can reasonably assume,
releases less dopamine in response to
an individual social interaction.
And so they need much more social interaction
in order to feel filled up by that interaction.
Now there's the fourth component
of this social homeostasis circuit that I mentioned before
and that's the prefrontal cortex.
The prefrontal cortex is involved in thinking
and planning and action and has extensive connections
with areas of the brain like the hypothalamus,
which is responsible for a lot of motivated drives.
It also has connections with the various reward centers
of the brain and it can act as kind of an accelerator,
meaning it can encourage more electrical activity
of other brain centers or as a break on those brain centers.
So while there are some predictable elements of these circuits,
they are not simply what we would call plug and chug.
You have flexibility.
You are able to say, you know, I love parties,
but I really don't wanna go to that party
because so and so is there.
So now I'd like to drill a little bit deeper
into this incredible neural structure
that is the dorsal raffae nucleus
and this small collection of neurons,
the dopamine neurons of the dorsal raffae,
because while it's a small collection,
they are very powerful.
And it is this dorsal raphae nucleus
and the dopamine neurons in that nucleus
that underlie the bond that is social friendship
and all types of social bonds.
There's a key finding in the literature.
The title of this paper is dorsal raphae dopamine neurons
represent the experience of social isolation.
This is a paper from KTai's lab.
What they did is they were able to selectively activate
the dopamine neurons in the dorsal raffey nucleus.
And when they did that,
they induced a loneliness,
like state. Now, how did they know it was a loneliness like state? They knew because it motivated
the seeking out of social connections. This is the kind of social hunger that I was referring to
before. Whereas when the dopamine neurons of the dorsal raffae are inhibited, meaning their activity
is quieted, that suppressed a loneliness state. So that's a little counterintuitive, right? It's a group
of neurons that when activated makes you feel lonely. And when this brain area is not
activated, it suppresses loneliness.
But if you think about it, that's exactly
the kind of circuit that you would want
in order to drive social behavior.
When you're feeling lonely, dopamine is released
and it causes you to go out and seek social interactions.
So what we think of as loneliness
as this big kind of dark cloud or fog
in our psychological landscape boils down
to a very small set of neurons releasing
a specific neurochemical for motivation.
So just a couple of key points
and actionable takeaways based on the information
I've offered up until now.
If you think of yourself as an introvert,
it's very likely that you get a lot of dopamine
from a few or minimal social interactions.
Whereas if you're an extrovert,
contrary to what you might think,
social interactions are not gonna flood your system with dopamine.
They actually are going to lead to less dopamine release
than it would for an introvert.
And therefore you're going to need a lot more social
interactions in order to feel filled up by those interactions. Now I'd like to shift gears a bit
and focus on what are some things that we can do to encourage the formation of healthy bonds.
There's a beautiful study that was published in Cell Report, Cell Press Journal, Excellent Journal.
The title of this paper is Conscious Processing of Narrative Stimony synchronizes heart rate between
individuals. Now, this study involved a very simple type of experiment. They had people listen to a
story. Everybody in the study listened to the same story, but they listen to that story at different
times and indeed in different locations. So different people, same story. And they measured things
like heart rate. They measured breathing, et cetera. Now, what was the motivation for doing this?
Well, there's a longstanding literature showing that our physiology, things like our heart rate,
our breathing, our skin conductance, meaning the amount of sweating can be synchronized between
individuals and that synchronization can occur according to a variety of different things.
What this study found was that when people listen to the same story but at different times,
their heart rates start to synchronize.
Now we also know from an extensive literature that the quality and perceived depth of a social
bond correlates very strongly with how much physiological synchronization there is between individuals.
In other words, when your bodies feel the same,
you tend to feel more bonded to somebody else.
And so this whole thing is a rather circular argument.
When you feel closer to somebody else,
your physiology synchronize.
And the reverse is true as well.
When your physiologies are synchronized,
you feel closer to other people.
This really points to the fact
that the body and the brain are reciprocally connected.
Yes, indeed, what we think, what we hear, what we feel,
drives our physiology, our heartbeat, our respiration,
but our heartbeat and respiration also
are influencing our state of mind.
And in this case, it's encouraging certain types
of social bonds when our heart rates are synchronized.
How can you leverage this?
Well, many people, when they interact with others,
expect that the mere interaction with the other person
is going to create the sense of bonding.
But in many types of social interactions,
it's not the direct,
the direct interaction with that person
that makes us feel close to them,
but rather it's shared experience.
And shared experience is shared physiology.
This is actually at the seat of what we come away
from a social interaction with as feeling,
wow, that was a really wonderful time.
Often a really wonderful time can be by virtue
of the specific things that were said
or the specific things that one engaged in,
but more often than not,
the final common pathway, we should say,
of great experiences,
was a great physiological experience
and a shared physiological experience.
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Up until now, we've been talking about social bonding through the lens of neural circuits
that are already established.
However, these very neural circuits that are responsible for social bonding in adult forms
of attachment, be it romantic or friendship or otherwise, are actually established during
development.
One of the more important and I think exciting areas
of early attachment as it relates to adult attachment
comes to us from the work of Alan Shore.
Alan Shore spelled A-L-L-L-A-N-S-C-H-O-R-E is a psychoanalyst
who also has deep understanding
of neurobiology of attachment,
both in childhood and in adulthood.
And he's focused a lot on differences
between right brain
and left brain forms of attachment.
Now, in a early episode of the Huberman Lab podcast,
I touched into the fact that most of what's discussed
in the general public and sort of pop psychology
and even in some neurobiology courses
about right brain versus left brain
and one side of the brain being more emotional
and the other side being more rational
is completely wrong.
However, the work of Alan Shore
points to some very concrete neural circuits
that do have a lateralization,
bias, meaning they are more right-brain than left-brain or more left-brain than right-brain
that underlie certain forms of attachment between child and parent, in particular, child and
mother, and that these right-brainisms, if you will, and left-brainisms for attachment
get played out again and again in our forms of attachment as adults. So within the field of
psychoanalysis, there's a long-standing discussion, of course, about the so-called unconscious
or subconscious, the things that we are not aware of.
And I think there's growing evidence pointing to the fact that at least one major
component of the subconscious or the unconscious is the so-called autonomic nervous system.
The autonomic nervous system is the portion of our nervous system that controls our
reflexive breathing, our heart rate, our skin conductance, meaning our sweating, pupil size.
It's the aspect of our nervous system that makes us more alert or more calm.
It's the so-called sympathetic, meaning for alertness or parasympathetic.
branch of the autonomic nervous system,
parasympathetic for more calming responses.
Now, what Dr. Schor's work and the work of others is now showing
is that early infant parent, in particular infant mother attachment,
involves a coordination or synchronization of these right brain circuits
and these left brain circuits as they relate, excuse me,
to the autonomic nervous system.
How does this play out?
Well, it plays out where early on,
as an infant, when you're born, you're truly helpless.
You can't feed yourself, you can't warm yourself,
you can't change yourself, and you certainly can't ambulate,
walk anywhere to get the things that you need.
All of those functions, all of those needs rather,
are met by your primary caretaker.
Typically, that's the mother.
I realize there are exceptions, but that's the general rule.
There are now brain imaging studies
examining the brains of infants and the brains of mothers
as they interact and showing that the physical
contact between the two, the breathing of the mother and child,
the heart rate of the mother and child,
and indeed the pupil size of the mother and child
are actually actively getting coordinated.
In other words, the mother is regulating
the infant's autonomic nervous system primarily,
and the infant is also regulating the mother's
autonomic nervous system.
A small coup from a baby or a cry,
which is a stress cry from a baby,
will definitely regulate the autonomic nervous system of the mother.
Now, as we get older,
there's another system that starts to come into play
in parent-child interactions,
and this also comes into play
in sibling interactions and so forth.
And that's the left brain system
as described by Alan Shore.
Now again, this isn't about emotion versus rationality.
This is about autonomic versus more conscious forms of bonding.
So on the left brain side of things,
there is a processing more of narratives
that are very concrete, logical narratives, okay?
And again, I have to zoom out
and just really,
tamp down the idea that it's not that one side of the brain is emotional and the other side
is rational, but rather there's a bit of a dominance for the left brain circuitry to be involved
in the kinds of bonding that are associated with prediction and reward. The idea is that there
are two parallel circuits that are important for establishing bonds and that this is set up very
early on in childhood and that it's neither emotional nor rational but both. What's becoming clear
from the neurobiological imaging studies
is that as people start to advance
into adolescence and adulthood
and well into their elderly years,
the same circuits that were active
and established in childhood
are repurposed for other forms of attachment
and that to have truly complete bonds
with other individuals,
but in particular with romantic partners,
it's important that there be
both synchronization of physiology
and synchronization
of these more, I guess we could call them
more rational or predictive type circuits.
So we can leverage this information.
In the clinical psychology and in the neurobiological literature now,
it's understood that there is both emotional empathy,
like actually feeling what somebody is feeling
and what is now called cognitive empathy.
Cognitive empathy is this idea that we both see
and experience something the same way at a mental level.
Emotional empathy is this idea
that, yes, I can feel what you feel at a visceral, somatic, or autonomic level.
And it's absolutely clear that strong social bonds between children and caretaker involve
both emotional empathy, this autonomic function, and cognitive empathy, that there's a mutual
understanding of how the other person feels and how the other person thinks in order to be
able to make predictions about what they're going to do. Now, it's also very clear,
based on the emerging literature, that romantic relationships and to some extent, friendships,
although friendships have been explored a bit less in the literature, that emotional empathy
and cognitive empathy are both required in order to establish what we call a trusting social bond.
So for those of you that are seeking to establish deeper bonds or bonds of any kind, it's important
that you think about synchronization of bodily states.
We talked about that earlier and synchronization of cognos states.
Now, that doesn't mean you have to agree on everything.
In fact, oftentimes people who feel very close to one another cognitively and emotionally
argue about all sorts of things and disagree about a lot of things.
But the point isn't that there be total convergence of opinion or stance, but rather that
we understand how the other feels and we believe that they understand how we feel,
that we understand how the other person thinks and that they think that we understand how
they think.
So it's a reciprocal loop between two people that involves this cognition.
and involves emotion and it's grounded as Dr. Shore has pointed out in our earliest forms
of attachment and that makes perfect sense because the same sorts of circuits that are
responsible for social homeostasis, the kind of right brain and left brain circuits that are
responsible for infant mother attachment and then later for more intellectual or predictive type
attachments between child and caregiver are the exact same circuits that we superimpose
into all other types of relationships
throughout the rest of our life.
And I should just mention that for those of you
that might be thinking that you had a less than satisfactory
infant caretaker interaction or form of attachment,
you are not alone.
And in fact, much of the work that Dr. Shore focuses on
is about how those early circumstances
can be understood and rewired
toward the development of healthy adult attachment.
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One of the key themes to understand
about biological processes is that they often work
on short timescales and longer timescales.
And up until now, we've mainly been talking
about the stuff that happens on short time scales.
So the kind of synchronization of heart rate,
or activation of a given set of neurons
that dumps some dopamine and causes us to seek out
more social interaction or less, for instance.
But every biological circuit and function
needs to have longstanding effects as well.
And typically when you're thinking about longstanding effects
in the brain and body, you start looking towards the hormone system.
So if ever there was a hormone or hormone-like molecule
that's associated with social bonding, it's oxytocin.
Oxytocin is involved in
Social recognition.
That's right.
When you see people that you consider your people,
your team, your group, your friends, oxytocin is released.
Even if you don't come into physical contact with them.
Oxytocin is also associated with pair bonding,
the feeling that they are your person
and that you are their person.
There's a common language people use.
It's also associated with honesty.
Believe it or not, there are experiments that show
that if people receive oxytocin
through an inhalation spray,
that they will be more honest and forthcoming
about certain things.
The main types of interactions that release oxytocin
at high levels are, first of all,
that the interaction be between individuals
that see each other as very closely associated, right?
Oftentimes they are in close contact,
oftentimes they are from the very body of the other.
And so the amount or the amplitude of oxytocin release
tends to scale with how closely associated individuals are.
Just the sight of one's baby
or smell of one's baby
can evoke oxytocin release and vice versa from the mother.
Physical contact even more so in romantic partners.
Physical contact, even the site of a picture of a partner
can evoke oxytocin release and sexual desire also trust.
And it's important to point out that that feeling of connection
is of the autonomic type that I was referring to earlier
a la Alan Shore's work.
That it's not of the, oh, we think about things
the exact same way, we agree on everything now.
It's more of that there's
physiologies are synchronized.
But the point here is that there's actually
a hormonal glue between individuals, okay?
Infant and mother, friends, teammates, romantic partners,
and so on, and that hormonal glue is oxytocin.
So we've covered a lot about the biology
and indeed the neural circuitry and neurochemistry
and neuroendocrinology of social bonding.
I wanna make sure that I highlight the key features
that go into any and all of your social bonds.
First of all, all social bonds have the potential
to include both what we called emotional empathy
and cognitive empathy.
And so if you are interested in establishing
and deepening social bonds of any kind,
it's important that you put some effort toward
this thing that we call emotional empathy,
which is really about sharing autonomic experience.
Emotional empathy and the synchronization
of autonomic function, heart rate breathing, et cetera,
can be best accomplished by paying attention
to external events, in particular narrative, story,
music, perhaps sports or other types of experience
as an external stimulus to drive synchrony
of those internal states.
The other aspect of forming deep bonds is cognitive empathy.
Again, cognitive empathy is not about agreeing on things
or viewing things the exact same way.
It's about really gaining understanding
of how somebody else thinks about something,
really paying attention to that
and then paying attention to how you think about
and feel about something.
Now, we also talked about introversion and extroversion.
And I'd like to try and dismantle the common misperceptions
about introversion and extroversion,
because when we look at the neural circuitry,
as you recall, introverts are not people
that don't like social interaction.
It's just that they feel filled up or sated
by less social interaction than would be an extrovert.
And that's because, at least according
to the social homeostasis circuit model,
they actually get more dose,
dopamine from less social interaction,
whereas extroverts get less dopamine release
from an equivalent amount of social interaction.
So for those of you that feel as if you're an introvert
or extrovert or that no introverts and extroverts,
it's not about how verbal people are.
It's not about how much they seek out social interactions per se.
It's about how much social interaction
is enough for the given person.
Now the whole reason for providing this framework,
this biological circuitry,
et cetera, is not to simply put a reductionist view
on things that you already realized and knew,
but rather to give you some leverage points
to understand how is it that you form social bonds,
how is it that you might be challenged
in forming certain types of social bonds
and to think about entry points
to both establishing and reinforcing social bonds
of different kinds.
Hopefully it will also give you insight into why breakups,
whether it be between friendships
or romantic partners, can be so painful.
A breakup of any kind of,
kind involves both a breaking of that emotional empathy
and that cognitive empathy.
And indeed, it has a neurobiological
and hormonal underpinning, right?
If one of our major sources of oxytocin
or one of our major sources of dopamine
suddenly is not around,
that is incredibly devastating to a nervous system.
And to borrow from the great psychologist
and neurobiologist Lisa Feldman Barrett,
who says, you know, we are not just individuals,
we are nervous systems influencing other nervous systems,
nervous systems and their nervous systems are influencing us,
I think that's the right way to think about it.
So it should come as no surprise that breakups of various kinds
are very challenging, regardless of what underlied
that breakup, whether or not somebody moving
or an actual decision of one person
to leave the relationship or both, et cetera.
Social bonds are vitally important to us as a species,
whether or not they are at a distance over social media,
whether or not they are in close proximity,
actual physical contact.
Today what I've really tried to illustrate
is that there are a common set of biological,
neurochemical, and hormonal underpinnings
to what we call social bonding.
And so while it is complex and it is subjective,
it involves the hierarchies,
it involves our previous upbringing,
it involves our goals, et cetera,
it is not infinitely complex.
And in that sense, it is tractable.
Hopefully I've offered you some levers
or some entry points under which you can both understand
and move towards social bonds
that would be more satisfying and more gratifying for you.
That's certainly one of the goals.
The other one is that hopefully if you're a clinician
or simply the friend that people go to
or the family member that people go to
when they are challenged through various challenges
and social bonds, that you can start to perhaps pass along
some of the information as a way of people
understanding what they're going through
as they are breaking up,
but also as they are falling in love,
as they are forming attachments
and as they are being challenged with attachments.
That's my hope.
And especially as you head into the holidays and end of year,
I would hope that you would take this knowledge
and apply it in any of the ways that you feel
are meaningful and adaptive for you.
