Ask Dr. Drew - Foot-long Blood Clots: Pathologist Dr. Ryan Cole Analyzes mRNA w/ Dr. Kelly Victory – Ask Dr. Drew – Episode 177

Episode Date: February 5, 2023

Dr. Ryan Cole returns for Part 2 of his mRNA analysis, and the adverse reactions that he believes are causing sudden adult deaths and foot-long blood clots. 「 WATCH PART 1: https://drdrew.com/11302022 」 As the owner of Cole Diagnostics, one of the largest independent diagnostics laboratories in Idaho, Dr. Cole alleges he has seen – and photographed – shockingly large blood clots appearing since 2021. And for patients who believe they’ve been injured by the shots, Dr. Cole has a simple recommendation: “Sue your doctor.” Find more about Dr. Ryan Cole at https://rcolemd.com 「 SPONSORED BY 」 • BIRCH GOLD - Don’t let your savings lose value. You can own physical gold and silver in a tax-sheltered retirement account, and Birch Gold will help you do it. Claim your free, no obligation info kit from Birch Gold at https://birchgold.com/drew • GENUCEL - Using a proprietary base formulated by a pharmacist, Genucel has created skincare that can dramatically improve the appearance of facial redness and under-eye puffiness. Genucel uses clinical levels of botanical extracts in their cruelty-free, natural, made-in-the-USA line of products. Get 10% off with promo code DREW at https://genucel.com/drew 「 MEDICAL NOTE 」 The CDC states that COVID-19 vaccines are safe, effective, and reduce your risk of severe illness. Hundreds of millions of people have received a COVID-19 vaccine, and serious adverse reactions are uncommon. Dr. Drew is a board-certified physician and Dr. Kelly Victory is a board-certified emergency specialist. Portions of this program will examine countervailing views on important medical issues. You should always consult your personal physician before making any decisions about your health.  「 ABOUT the SHOW 」 Ask Dr. Drew is produced by Kaleb Nation (https://kalebnation.com) and Susan Pinsky (https://twitter.com/firstladyoflove). This show is for entertainment and/or informational purposes only, and is not a substitute for medical advice, diagnosis, or treatment. 「 WITH DR. KELLY VICTORY 」 Dr. Kelly Victory MD is a board-certified trauma and emergency specialist with over 30 years of clinical experience. She served as CMO for Whole Health Management, delivering on-site healthcare services for Fortune 500 companies. She holds a BS from Duke University and her MD from the University of North Carolina. Follow her at https://earlycovidcare.org and https://twitter.com/DrKellyVictory. 「 GEAR PROVIDED BY 」 • BLUE MICS - Find your best sound at https://drdrew.com/blue • ELGATO - See how Elgato's lights transformed Dr. Drew's set: https://drdrew.com/sponsors/elgato/ 「 ABOUT DR. DREW 」 For over 30 years, Dr. Drew has answered questions and offered guidance to millions through popular shows like Celebrity Rehab (VH1), Dr. Drew On Call (HLN), Teen Mom OG (MTV), and the iconic radio show Loveline. Now, Dr. Drew is opening his phone lines to the world by streaming LIVE from his home studio. Watch all of Dr. Drew's latest shows at https://drdrew.tv Learn more about your ad choices. Visit megaphone.fm/adchoices

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please contact Connex Ontario at 1-866-531-2600 to speak to an advisor free of charge. BetMGM operates pursuant to an operating agreement with iGaming Ontario. Of course, today is the part two of Dr. Ryan Cole's appearance here. Dr. Cole is a Mayo Clinic trained board certified anatomic and clinical pathologist, subsequent fellowship in dermatopathology and a PhD in immunology and virology, currently CEO of Cole Diagnostics. And he has been one of the great sources

of the PATH specimens upon which we have been tracking some of the injuries, some of the injuries from COVID, some of the injuries from the vaccine, and trying to parse these things out. He has been at the front lines of it, and he has sort of been at the center of the spokes in terms of having access to a ton of material. And he's here to report, give us an update today. So, you were very excited about the last time he was on. We were very excited to see him this time. Dr. Ryan Cole, right after this. Our laws as it pertains to substances are draconian and bizarre.

A psychopath started this. He was an alcoholic because of social media and pornography, PTSD, love addiction, fentanyl and heroin. Ridiculous. I'm a doctor for f*** sake. Where the hell do you think I learned that? I'm just saying. You go to treatment before you kill people. I am a clinician.

I observe things about these chemicals. Let's just deal with what's real. We used to get these calls on Loveline all the time, educate adolescents and to prevent and to treat. You have trouble, you can't stop and you want help stopping, I can help. I got a lot to say. I got a lot more to say.

And as always, we'll be watching you on the Restream and the Rumble Rants. We appreciate you all being here. Tomorrow, we're going to switch gears a little bit and do a little bit sort of economics. We had a big report from the Federal Reserve. And we thought it might be interesting to have sort of a different point of view about sort of, you know, homo economicus, which is, you know, does overlap with some of medicine to some extent. All right. So Dr. Kelly Victory, of course, will be with us here in just a few moments. But let's go ahead and bring Dr. Ryan Cole in. Welcome, Dr. Cole. Thank you so much for being here again. Hey, great to be here again. Like last time, I didn't finish my PhD. I was an MD-PhD

candidate and had my first baby on the way. So I did my immunology, virology research, and at that point, went first baby. So I catch grief for everything. I'm one of the favorite people to attack on the internet. So I just want to make that that clear i am an md board absolutely no no i appreciate that because i do not want to get that all wrong we want to present our you know it's so it's so this is just a complete sidebar but it's astonishing to me that people who claim to be experts in all kinds of fields you never can find their training magically or you're never clear what what it is that they are you know how they're in a position to say the things they say for instance and uh boy i've

noticed it over and over again when they when the training is not right up front there's a reason for it those of us that have had lots of training a lot to experience we want to make it clear exactly what our training has been and you know you know, just, you know, because it takes decades to do this stuff. And we want you to know that's what we did to be able to have the judgment to talk about things we're talking about. All right, so let's talk about what we're talking about. Do me a favor, if you wouldn't mind, Dr. Cole,

and just sort of, let's reset, just in case there are people here who didn't see your last appearance. I don't want them to have to go necessarily back and listen again, although they certainly are willing to. Just how you got into this, what you're seeing, and then now what you're seeing at the present moment. Certainly. So from the beginning of COVID,

having an interest in virology and having an interest in all things pathology, did a deep dive, was very familiar with coronaviruses from previous study. And so when community, local hospitals, and made sure that we were playing a pivotal part to help our community get through what was coming. And early on, I was a big skeptic of a program for injections, vaccines against coronaviruses, just understanding how coronaviruses mutate and whatnot. So I was highly concerned there at the beginning, highly concerned about the experimental technology being used, the lipid nanoparticle plus a gene sequence had never been used widespread on humanity before.

So that had kind of my radar up watching for what the effects might be. And certainly here in the laboratory over COVID, we certainly saw certain patterns that presented themselves. And if anybody wants to go back, you and I discussed that a lot of COVID was clotting and endothelitis, inflammation of the blood vessels. And so I was seeing some of that clotting under the microscope during the first year of the pandemic before the shots rolled out and certainly seeing more of it after they rolled out. And so that's part of what I've been doing is studying the different patterns in the laboratory.

And what a pathologist does is we're the quality control of medicine. We look for patterns, changes in patterns, and that helps inform clinical medicine. And so there are different techniques we use in the laboratory. Certainly there are basic things that we've been doing all along, looking at white blood

cell counts and certain counts going down after COVID and most certainly going down after the injections as well. So just basically following patterns and then trying to, in a voice of just scientific reason and trying to keep it reasonably down the middle, say, look, here are the patterns we're observing. We should be concerned about these. We should study these things. And that's kind of where we are now. We've advanced in terms of how we can identify effects of the virus, effects of the vaccine, both within the human body, within the tissues, within the cells. And as I like to say,

the cells don't lie. They're objective. And the patterns that we see in those cells inform us even more, you know, what we've done right, what we've done wrong, and what we could do better in the future. And so that's basically what's brought us, you know, to where we are now. You and I covered a lot of these different issues, and I'll present a few of the slides from the previous presentation again today, just for those who didn't see it. And then we'll go over some newer findings that are, again, still becoming very concerning. And hopefully, you know, not to alarm or scare anyone, but to simply give people an idea of what we do know, what we don't know. And at the end of the day, have hope for those who may be still suffering long COVID,

those who may be suffering a vaccine injury, and find some of these pathways and mechanisms and pathology so we have answers to help people that are in those unfortunate scenarios. The question I keep asking is, you know, how do, I'm trying to think in terms of rendering really informed consent to my patients that are younger. How do we understand, and what is your sense of the incidence of, let's just take clotting for insights. I'm not sure we should take one particular pathology, but I can't think of a better way to frame this. Take clotting, what's COVID, what's vaccine, what's vaccine plus COVID. How do we adjudicate the risks?

Yeah. And unfortunately, we've layered so many of those things societally on top of each other that teasing those out from each other becomes a challenge because obviously early on, even if you were COVID recovered, and a lot of people had COVID, didn't know they had it, and then were encouraged to get a shot on top of their COVID recovery, their natural COVID recovered immunity, which generally historically we don't do. And even Fauci in 2018 said, look, the best vaccination of all is recovery from a disease when he was talking about flu. So we know historically in immunology, the immune system does a fantastic job, especially if one isn't decent health, normal vitamin D levels help maintain memory responses in the

immune system and whatnot. So to try to tease out, and it's interesting too because if you go back and look at say some of the Moderna studies, if you got two shots and then ended up with COVID, your immune response was actually weakened and blunted in certain areas. That was week 42 data out of the United Kingdom. So there are things that we've done that haven't been very scientific as we've gone along. And this has been a frustration for me

is finding those cohorts that are pure, those that were unvaccinated, COVID recovered, and looking at their data sets and then comparing those to, and, we do have 30%, at least here in North America, that never got the shot. So we do have a fairly good cohort here. But then looking at those who had COVID, then got a shot, and then got COVID again, that alters some of these patterns. And so there's not any one good percentage. The clotting, I really go back to the beginning of the pandemic. And in my mind, primarily, secondarily, tertiarily, COVID is a clotting disease. And we can look, there was a large poll done that showed out of 100 Americans post-injection,

15% ended up with a new chronic condition. So what that tells me, if we some year, two, three, five years from now have the data sets, we can genetically go back and say, okay, who was susceptible and why? Why did the spike protein affect this subset of people so adversely compared to those who got COVID or got a shot and had no problems? We don't have the answer. We have the technique. we have the means. Do we have the political will

to put the finances into those areas? We spent plenty of money advertising a new modality, but those same billions of dollars should have gone into the research at the same time and beforehand to understand what would happen from an immunologic basis, from a clotting basis, from a tissue basis, from all these findings that we're seeing. So I think that's the big picture is it's muddy. Yeah. And I know you've been very focused on the spike protein and

its effect on clotting and endothelial function. Is there any, before I bring Dr. Victory in here, I want to know if you have any thoughts about the liquid nanoparticles and could they be a part of the problem as well? And have you had anything on that lately? I don't think we really talked about that last time. Not specifically. Yeah, this is probably one of my biggest concerns of all is a lipid nanoparticle. These were designed to carry chemotherapeutic agents to the brain. So we know that they cross the blood-brain barrier. We know lipid nanoparticles go everywhere. The biodistribution studies, and I know you had Dr. Breidel on from Canada, the biodistribution studies that were done out of Japan show how widespread this lipid nanoparticle is within

the tissues. And so that's a concern. Human cells were meant to make human proteins, not foreign proteins. So when this lipid nanoparticle carries a gene anywhere in the body, now those cells can express the protein that that gene codes for. So what we do know in retrospect now, the damage that the spike protein can cause, my biggest concern is the industry seems to think they have carte blanche going forward now with this new platform of a lipid nanoparticle and a gene sequence for RSV, for influenza, for

whatever other pathogen. And the problem therein is we don't have long-term safety studies. Usually a vaccine takes six, 10 plus years to bring to market because we want to see all those safety signals, tease them out and make sure it's safe. It doesn't matter if it's effective, if it's not safe. And so we had all these upfront claims of efficacy, even though those were based on relative risk and not on absolute risk reduction. So the problem is we have a delivery platform that's never widespread been used in humanity, which had shown problems. The lipid nanoparticle in and of itself is very inflammatory.

And those lipid nanoparticles, because they're positively charged, they'll fuse and intercalate into the cell membranes. And so when we studied these, we didn't study these in multiple dosages. Initially, studies on lipid nanoparticles were single dose. And that was the promise of it for inborn errors of metabolism, genetic disorders, and whatnot. You could quickly deliver a gene for those missing genes in those kiddos or in certain conditions everywhere. Well, that's great for rare conditions, but for vaccinal technology, that has implications when it goes to your liver, to your bone marrow, to your spleen, to your brain, to your heart, to every organ in the body, the adrenal are expressing glee, hey, look, we can use this for all these other types of pathogens. Gosh, you know, the precautionary principle in my mind should take precedence here. It's really disturbing and concerning that the industry is, well,

and they've kind of been given a green light by the regulatory agencies, unfortunately, when we don't have the safety data. And even to that point, these were authorized as though they were regular protein-based type vaccines and under the regulatory statutes of vaccines and not under gene-based products. And these are gene-based products. And all those required studies that should be done, the reproductive toxicity, the mutation toxicity, the dose-dependent toxicity, et cetera, those weren't done. And so we have something being called a vaccine, but it's a gene-based product, and that's highly concerning.

Now, we also have a clotting update for us. Is that the PAI1 inhibitor? Is that part of that update? Tell us about that. Yeah. Okay. So, you know, you and I were kind of talking beforehand a little bit about going back to med school and remembering all these clotting cascades. If I could have Caleb jump real quickly to slide number 19. So when you clot, there's a lot of little factors that basically a waterfall, a cascade of events that have to happen to form a clot, which is great because if you have a scrape on the knee, you obviously want that to stop bleeding. You want a little natural bandaid from inside your body to patch things up so you don't ooze outside yourself. You basically want a patch. Well, once a clot has formed, then the body has to break down that clot and kind of remodel

that area as your scar starts to form, you start to heal. There are certain things that happen to have to happen sequentially as well for these clots to break down as well. And what we're finding, and I give Dr. Raisa Pretorius out of South Africa, you know, grand kudos. She's been on the leading edge of studying this with COVID as well as from the injections. And so has my colleague down in Alabama, Dr. Jordan Walker. So I borrowed his slide here.

So this plasminogen activator inhibitor, if you have a gene mutation for this, now you're not able to properly break down those clots. And in addition, if you have a gene, there's a 4G and a 5G, and you can either have one copy or two copies. And in these scenarios, if you have this plasminogen activator inhibitor deficiency, now you can't break down clots. And there's a subset of the population, bump to the next slide, Caleb, a subset of the population where it becomes concerning. When you have these microclots and you can't break them down, now your oxygen can't get to your tissues properly so ischemia is lack of oxygen to those tissues uh as well as hypoxia which can lead to fatigue right good and so so one of the things that one of the couple things uh do you ever see a larger

cascade and developing into a larger clot and one of of the observations, say, on what's causing people to lose their sense of smell and whatnot, and certainly what's probably still making my right ear ring, is microvascular changes in the CNS. And I'm wondering if those are clot-based also. So bigger clots and very tiny clots. And are the microvascular, microvasculitis or whatever it is, is it primarily a clotting problem? I think, again, this will go back to, you know, the previous show where you and I hit on this. I think the primary process in COVID spike protein injury is an endotheliitis, the inflammation of those vessels. And once it's inflamed, once the vessel lining is inflamed, then you end up with these micro clotting cascades. And so it's interesting societally,

prevalence of this inhibitor, you see it more in obesity, you see it more in diabetes. Obviously, societally, we have a very metabolically unwell society, a lot of insulin resistance within our society from our processed foods and unhealthy, I call it our SAD, our standard American diet. And around the world, hopefully don't follow our example. But when you have those pre-existing conditions, these kind of things do increase. And I agree with you i think some of those sequelae some of those you know post-covid happens post-injection things are because some people are forming microclots and not breaking them down yeah right and so if you are one of those people with the

i'm going to call it genetic deficiency it's really just a unique genetic profile around plasminogen activators uh while in normal balance, they're okay. It's when something causes it to go out of balance that it really gets bad, right? Or something is happening systemically. But my question is, is there an association between those genetic subtypes and what we're seeing in the cytokine system? Has anybody made that link? Yeah. And interestingly, so the more adipose one carries on their body, you're already in a pre-inflamed state. So interleukin-6 is one of the inflammatory cytokines. So unfortunately, we saw the outcomes and the data outcomes, and that was the cohort that did the worst was the increased adipose.

But is that linked to these same genetic folks with their plasminogen? Yeah. Okay. It is. And not just from a, oh, you've got a genetic disorder point of view, but rather these types of fact are affected by those metabolic factors. So you're absolutely right. You can change your gene expression profile based on your underlying health. So you change your underlying health and some of these activators will go back to more that balanced homeostasis got it all right dr cole let's do this let's take a little break i want to bring uh dr victory in with us she might have to leave us a little early so i'm going to bring her in as quickly as possible here so we'll take a little break uh i'll also watch for your questions

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Birch Gold has an A plus rating with the Better Business Bureau, countless five-star reviews and thousands of satisfied customers. Check them out now. Visit birchgold.com slash Drew and secure your future with gold. Do it now. Some platforms have banned the discussion of controversial topics. This episode ends here. The rest of the show is available at drdrew.tv. There's nothing in medicine that doesn't boil down to a risk-benefit calculation. It is the mandate, public health, to consider the impact of any particular mitigation scheme on the entire population. This is uncharted territory, Drew. Hey there. How are you you ryan great to see you and uh thanks again for

joining us it's my pleasure great to see you again kelly terrific um lots i want to talk to you about but i think it bears repeating um did you want to do it sorry guys i was busy i was busy with the restream i was answering so many questions i got deeply into it so they have lots of questions for you dr cole so go ahead kelly sorry for not introducing you properly that was a first no no no worries no worries um there are things i think bear repeating and one of which is that you and I have been flying in the same circle since the beginning of this pandemic. And you were the one person who I knew who was really bringing some explanation from a pathology standpoint to the things we were seeing clinically. We knew we were seeing blood clots, but you were able to show why. We knew we were seeing increases in infertility, but you were able to show why. We knew we were seeing increases in infertility,

but you were able to show why at a cellular level by looking at these actual tissues. And I think it's critically important for people to understand that the things we are seeing clinically are being proven, are being explained in what you are seeing at the cellular level under the microscope. And that's really, really important. One of the things I heard you say that I hadn't heard in our last conversation, and I think I want to make sure I understand it. You believe that there is a genetic predisposition or some genetic explanation for why some people are being harmed when others aren't. One of the things we've known from the beginning is that the adverse events, serious adverse events from these shots

are not distributed homogeneously. They appear to be different based on lots of the vaccines. So there's this whole, you know, how bad was my batch and the fact that you know like 80 some percent of the really serious events seem to be associated with a relatively small percentage of the vaccine lots so now i'm hearing a sort of a different take on this that maybe there's a genetic predisposition in individuals to be harmed so how have you figured out a way in your own mind to to bring those two things together that one appears to be variations in lots and the other appears to be an actual genetic predisposition to being injured? Great question, and I think it's both. Before I jump in, I want to answer from the previous segment.

I said decrease in PAA1. I meant increase in PAA1. So if anyone criticizes me on this previous segment, okay, go into your question. In the vaccine injury group, I agree there may be a higher concentration. There's a wonderful group of physicists out of Austria. I got to present their data. And there's inconsistency within the batches, most certainly. Some have more polyethylene glycol, some have less. Some have more lipid nanoparticles, some have less.

Are there contaminants? You bet there are contaminants. So the consistency of production may account for some of the injury that we are seeing. Now we do know genetically, and there was a really big study, I want to say it was a group out of Qatar that did some in silico modeling of the affinity for the spike protein to the ACE2 receptor amongst different racial groups. So interestingly enough, there is a genetic predisposition as to where that spike will bind depending on one's genetic

makeup. So, you know, a small amount of spike binding more strongly to some of those genetic groups will lead to, you know, more inflammatory outcomes. So we already know from an ACE2 receptor where that spike protein binds point of view that that's actually a shown factor. So I think it's a one-two punch. So number one, you get that affinity of binding. I think that's why certain cohorts did a little worse in terms of COVID outcomes, as well as showing vaccine injury susceptibility. But in addition to that, absolutely, the inconsistency, especially in the first couple

rounds. Now, I haven't been able to examine any of the bivalence or work with any of the physicists on that, but I do think that both of them play together in terms of those outcomes. And again, this is going to be something we're going to have to tease out. The key to science, there's two things that make up a good doctor. Number one, you care, and number two, you're curious. And if you don't have that curiosity, we'll never answer these questions. And the only reason I've been doing this and putting my neck on the chopping block trying to share science is I care. And I'll never stop being curious.

That's that's how we answer the questions. If we've done something that's harmed people, well, let's see if we can reverse that if we want to understand who's most susceptible, then we need to study these things. There are databases and there are large university systems that should be able to get together and tease some of this data out. I'm an independent laboratory. I can't do all these things. And a lot of these things cost money. Everything I've done so far and presented so far has been out of my own back pocket just because I care. I want the scientific answers so that we can prevent future harms. Well, and I agree with you wholeheartedly, and this is what I've said from the beginning, that we have got to care about this. Fundamentally,

these are things that, as you said previously, would have been sorted out, or many of these things would have been sorted out in the six to eight years that we normally take during the testing period for vaccines. There is a reason, vaccinology is complicated stuff. I have said for a long time that the immune system is the last great frontier of medicine. It is complicated stuff. The immune system doesn't always respond the way we think it's going to. And it's why many vaccines never make it out of the starting blocks. They fail and never get there. So I appreciate that. And it sounds like it's, you believe that it's probably multifaceted, both some components that are genetic and some that are related to the gross inconsistencies

in batches. Now, back to these blood clots, too. One of the things we talked about when you were on with us last was that the blood clots, in addition to being very massive, they also included, if I recall, a fair amount of amyloid, something that is not in normal blood clots. So let's talk about that. These aren't just, for those people who might not have seen part one, that these blood clots are not typical in that way, not just the size and the

locations and the frequency of them, but that they include some other components that you normally don't see in blood clots. Yeah. And this is a great point. I'm glad you bring it up. I'll have Caleb pull up slide number 15, if he would please. Here on the left, you can see there's a, there we go. The criticism that's bantied about is, well, gosh, you know, these are just postmortem and they're an incidental finding postmortem. And the clots you see on the right, yeah, those are postmortem clots. In fact, I have some spike stains coming here in about an hour from one of my technicians that will be looking at some of those with spike stains. But on the left, that was removed from a patient in vivo. I have other slides of patients that have like the entire bronchial tree, like it

was cast. One was a pilot, the other was an internal medicine doc in Florida. And so these large clots, they're unusual in the sense that they have a lot of proteinaceous deposition. And so if we go back to the conversation in the earlier segment with Dr. Drew, some of this clotting process is because excess proteins are being deposited and not broken down by the body. So if we hop to the next slide, Caleb, here you can see spike protein within a lung vessel. This is Dr. Burkhart's slide out of Germany. I've been working with him and communicating with him. Just saw him at a great meeting in Stockholm where we compared notes and are continuing to do these studies across the world together.

So here you can see spike protein in some of these clots. And if you go to the next slide, this is one from a patient that I just did, unfortunately, and this was a COVID patient, not a vaccine patient. But here you can see the spike protein, all that little green glowing stuff, that's micro amyloid deposition in capillaries within a heart. So the amyloid is depositing in vessel walls, and then some of these amyloid clots will piggyback and just basically like daisy chain onto each other. They just grow and grow and grow. And again, certain patients have the ability to break down clots and other patients are missing certain factors. And so it's, again, it's not known who is and who isn't. And sometimes we have to go on symptomatology.

But here's what's interesting. If we jump to the next one, we now have in the laboratory, and there's only a handful of labs doing this, on the left, we can stain with a very simple stain called thioflavin, and it'll show clumped protein. So if I take just a simple tube of blood, you know, if you go to your doc, get a tube of blood drawn, spin it down, take the serum off the top, and then we stain it with thioflavin. Now we can see protein clumps. Now we know which of these patients are forming microclots. And then on the right,

this is a chart from Dr. Pretorius in South Africa, again, showing that even in the absence of platelets, if you just put the spike protein in, the proteins clump. So, that spike protein induces clumping. Now, amyloid, the problem with amyloid is the body really doesn't have an easy way to break it down. What some of these docs are finding in their studies, if they put them on a triple anticoagulant therapy, they're finding a lot of these patients improve. I think you and I maybe hit on the enzyme natokinase when we were chatting last time, which is an alternate enzyme that also has, you know, fibrin breakdown effects. It's a fibrinolytic. So those who have symptomatology and some of these patients, again, I'm not your doctor.

This is just educational. But as a supplement, some patients are finding that natokinase is improving some of their symptoms. Not only does it inhibit spike protein binding, or in one study showed some spike protein degradation ability, but also inhibits clotting as well. So amyloid is a very concerning finding, and a lot of these are just microfibrils, like that little green glowing dot we see on the left, but some of this can lead to macro clumping. And that's where I think we're seeing the formation of some of the larger clots. And I think, you know, long COVID and vaccine injury, it's not all the same pathway. Some of

it's mass cell activation and allergy activation. Others are reactivated virus. I have a whole presentation I've done where the doctor can go through and say, okay, here's the four primary pathways of injury to explore. Obviously, one pathway is the clotting that we're discussing. And then mitochondrial harm is another major concern I have. And mitochondria, they're the little engines that drive every cell in your body and produce energy and we found in some studies around the world mitochondria are inhibited by the spike protein both from the infect so there's so many pathways but I think clotting is one

that should always be on the mind symptom or that logically because you can do something about it this is something you know we've been treating clotting for ages. And when COVID came along and in the hospitals, they were unwilling to just use simple anticoagulants. It was somewhat perplexing. Well, this is a good segue because we're talking about the staining, Ryan.

And one of the things that I know we talked about last show, but it's really, I think, critical, is this concept that we have, you have at your hands, the ability to stain and differentiate between spike proteins that have occurred as a result of the virus. We know that some of these things, some of these syndromes and some of these complications occur in people who've had COVID and who have long COVID. But you are able to differentiate at the cellular level, at the tissue level between spike proteins that occurred as the result of someone having COVID-19 versus

spike proteins that were produced in response to the mRNA that they were injected with in the vaccines. This to me is a critically important issue because it really answers the question, well, does this person have myocarditis or does this person have inflammation of the vessel wall because of these spike proteins? Where did those spike proteins come from? Did they come from that person having COVID the virus, or did they come from receiving an mRNA injection that induced them to create those spike proteins? So talk about how you make that differentiation. Dr. Certainly. I'll have Caleb jump to slide number three, and I'm going to jump through a couple slides really quickly here, Caleb. So when we got the shot, you see a

superimposed needle, again, Dr. Burkhart's slide from Germany. And you see in the deltoid muscle, that's spike protein being expressed. Now we were told that it would stay in the arm. Plenty of studies now show that it doesn't. We'll go to the next slide. This study was done by Dr. Roltgen et al out of Stanford. And on the left, you see mRNA persisting in lymph nodes for at least two months. And then on the right, the little brown dots, and this is what we look at under the microscope. These are all cells you see up to 60 days later, there's still spike protein being produced. We'll jump a slide forward. Is that new info, Dr. Cole? Is that new information that sounds new?

Unfortunately, no. I mean, this came out last year and there's several studies showing the persistence of circulating spike protein. There was one that came out of Harvard on myocarditis showing that the kiddos that got myocarditis had circulating spike compared to those who didn't get myocarditis. Six months is a new number to me. I had not heard six months. It was 60 days, I'm sorry. That was 60 days. And at 60 days, she took that to public. Okay.

But they stopped studying it at 60 days, if I don't recall, right? Yeah. That's when they stopped. I'm getting a little confused, guys. Is that, are we talking about the mRNA or the spike? Which are we talking about? Post-injection, Post-injection. Post-injection.

So these are post-mRNA injections. But persistence of mRNA or persistence of spike protein? Because I did hear you say 60 days, two months for the mRNA to persist. But then I thought I heard you say six months for the spike to persist. And that was like sort of mind-blowing to me. Six months. Well, here's here's what okay i will blow your mind okay so the mrna we know persists for at least two months in some

patients so does the spike for 60 days 60 days in that study 60 days spike in that study dr bonsal showed uh circulating spike uh four months later in the journal of immunology dr bruce patterson has shown circulating spike in in non-classical monocytes in the blood up to 15 months later. So we know that spike protein can stay in circulation for over a year. And we don't know when the mRNA breaks down. Now, this isn't in every patient. It goes back to Kelly's earlier point. But we don't know who has, because of the synthetic nature of this RNA, you know, some people are deficient in the ability to break down RNA to begin with. And it goes back to the

same groups you and I were talking about, Drew, obese, diabetic, et cetera, and certain racial groups. So some patients- Can I just, I want to flush out the Patterson's observation, because I'm very familiar with that. what they observed was persistent spike protein in non-classical monocytes in the central nervous system who typically go through a cycle of apoptosis very quickly. And what he's been able to show is that these spike-infected monocytes don't go through that cycle. They stay whole, and they do their inflammatory thing and they CNS and associated with elevated VEGF. Absolutely correct. Absolutely correct. And that, you know, I'm glad you're familiar with that because that's, I think that's societally the concern in these patients that are suffering long-term either post COVID and or post injection is the persistence in some of these individuals

within certain cell types. And to your point, that chronic inflammation that that induces will lead to the symptomatology that we're trying to resolve. So going to Kelly's question, so now we'll jump forward. Let's see. Let's jump forward two slides to number six, Caleb. So I'll go back one, and then this one's kind of a teaser because this one's important. So spike protein here on the left, this is heart tissue. All that brown that you see is spike protein by a special technique we use in the laboratory.

Again, this one's Dr. Burkhart's slide. The brown is spike protein expression and the antibodies we use in the laboratory are like a lock and a key. If it binds and locks on, then we use a little glow on the end of that antibody so we can see it under the microscope. So on the left, we have spike protein as an internal control. If it were a viral infection, then the nucleocapsid should light up as well. So it's simple deductive reasoning.

If spike is there, nucleocapsid isn't. It's not from an infection. It's because the body's producing spike protein. Now, if we jump to the next one, this is highly concerning. This is one I just completed yesterday on an individual. And this one we'll do more of a prominent press conference on once the rest of the autopsy is complete. This is the adrenal gland.

Your adrenal glands are so critical for so many functions in your body, from your adrenaline to your glucocorticoids to your hormone balance, even to your happy hormones, your metencephalins, et cetera. This individual, you can see all those brown dots in there. And again, the nucleocapsid was negative, and this was a post-vaccine death. And all those brown dots, that spike protein protein not only filling the middle of the adrenal gland but it was also in the medulla in the cortex as well so it was filling the entire adrenal gland what does this mean well a lot of patients have POT syndrome postural orthostatic tachycardia syndrome after COVID as well as after the injections. Well, this shows that it's

absolutely depositing in the adrenal glands. And so this is kind of a newer finding that I wanted to bring out here because so many patients are going through that post-COVID or that post-injection challenge of blood pressure regulation, of fatigue, et cetera. And so this one, I mean, hit me over the head when I saw this one yesterday. I'm like, oh my gosh, look at the amount of spike protein within the adrenal gland. And not just in one area of the adrenal, but distributed throughout. And if we take a step back, again, it explains some of the clinical manifestation that we need to be trying to address. Number one, can we neutralize the spike in these patients that are persisting with spike expression?

Number two, can we stop doing this to people? It's causing harms that we didn't study beforehand. And again, good medicine, it doesn't matter if it's effective, if it's not safe. And we're finding so many findings through the tissues. And as Kelly said, the cells are objective. That's what I like about pathology. It's, here's the observation. The cells don't lie. It's either present or not present. It's binding or not binding. And these cells are expressing it. And so we know from a pathophysiologic mechanism,

the cells are expressing spike protein. And that lipid nanoparticle, the adrenal gland is really fatty. And so when we were discussing before, Drew, the lipid nanoparticle honing to wherever it's going to hone to, it likes to hone to fatty tissues. And the adrenal gland has ACE2 receptors, but it's also a fatty type tissue. So here we can see a manifestation of it fusing, put its gene product in, and now these cells that shouldn't be making a foreign protein, making a foreign protein. And think of like Addison's crisis. That's when somebody's,

you know, blood pressure will drop because you don't have the appropriate hormones to keep tone in your blood vessels, can cause low blood sugar very quickly as well, can cause a dump in your potassium and your potassium goes low as well. Now even if there's no inflammation in the heart, if your adrenal gland isn't functioning properly, that could lead to an arrhythmia because of potassium insufficiency as well. So this is one new mechanism that just literally came to me yesterday as I was looking through slides and I have in the background here about a kajillion more to look at and those will uh what part of the adrenal gland are they is it is it showing in all parts the adrenal so you're getting mineralocorticoid as

well as glucocorticoid yeah yeah yeah the medulla and the cortex and as well as um yeah go ahead as you know as you say one of the things that that that you said Ryan that I really you know Yeah, go ahead. and we have got to focus on how to fix what has been done to these people. You just used the word, I think, you used a different word than I've said. Yeah, I've been figuring, saying, how can we remove these spike proteins? There's got to be a way to diffuse them or to remove them. I don't know how, do we electrophorese them out? Do we chelate them out? How do you get rid of them? Or, and, or how do we turn off this mRNA? I'm not an mRNA scientist. I don't know a lot about how it gets turned on, but if it got turned on, maybe there's a way to turn it off.

But to me, these are the things that we as scientists need to be focusing on. How do we get rid of this spike protein and how do we stop people from producing more of it? Yeah. And there are plenty of groups out there looking at this and there's plenty of claims out there. And I think some appear to be useful and successful to a degree. I've seen a lot of patients do quite well. One of the primary ways that I like with the FLCCC approach is autophagy. And that you induce through intermittent fasting. An intermittent fast is 16 hours. And then you cause some of these cells that are carrying atypical proteins or these cells that are weakened to die off so you can grow new healthy cells. So that's a simple

way is just cutting caloric consumption several times a week for 16 hours a day. I do that intentionally about four or five days a week, accidentally one or two days a week. But, you know, that's one amazing way to maintain health. The real answer is we don't know. And this is where I wish the studies were done to show when does the spike protein stop being produced and when does the mRNA break down? And these are tissue studies that should have been done in mammals to say, hey, this pseudoyurthine within this synthetic mRNA, we don't know the answer. That would be, gosh, the easiest study to do at any large lab, university level, et

cetera, and should be done because people need this answer. The answer to the question is, I know plenty of things that are useful, certain drugs that shall not be named, as we like to say, that do have spike inhibitory effects. The mRNA question is an unanswered question. I don't know. Before the clock runs down, there's something else I really want to pick your brain about. We've talked a lot about cardiac injury, and certainly that's very important. We've talked a lot about autoimmune issues and increases in cancer rates. One thing that I don't think has gotten enough attention in the mainstream so far are the neurologic or psychologics, you know, neuropsychiatric issues. Let's switch for a little bit and talk about, we know based on DMED, the Defense Military Epidemiology Database, and from claims that there's a significant increase in new onset seizures. We're seeing people complaining of this brain fog, the persistent brain fog, different neurologic problems. Let's talk again at the

cellular level from what you're seeing, the pathology. What are you seeing in brain tissue that could potentially explain some of these things? All right. Number one, I'm going to have Caleb jump to slide number eight. This is a case that I received recently of a deceased individual, unfortunately. The spike protein has the ability to fold in interesting ways. So certainly, we have that amyloid question. And amyloid, a certain type of amyloid is associated with Alzheimer's. I have a colleague in California that runs a memory care clinic. After the shots rolled out, she saw a massive decline in function within her elderly patients.

Now, this one that I'm showing right here, Dr. Montagnier, the Nobel Prize winner, he had identified 26 cases post-vaccine of spongiform encephalopathy. People are familiar with the term mad cow disease. This is a case, all those white holes that you see, that's why they call it spongiform. It looks like a sponge. So this is an individual who unfortunately passed away shortly after his third shot, and his brain was Swiss cheese. And not all of it, but good parts of it and you can't have cellular function if the cells are blown apart now if we'll jump to slide 10 here on the left this is a single case studied by dr. Moore's on the left that showed again like dr. drew mentions inflammation of the vessels all that

brown on that little tube on the left that spike protein then you see leaking out from that vessel little brown dots into the surrounding brain tissue. Now on the right-hand side, these are slides I've completed. These are from that same patient that I was showing you with the sponge-like pattern in the brain. Those brown dots in the middle, those are neurons, and those neurons are being rimmed with spike protein. And so we know the spike protein crosses the blood brain barrier, the S1 fragment. We know that the lipid nanoparticle crosses the blood brain barrier. So unfortunately, that's a very protected space in the body. It's an immune protected site. So to see the pattern of a protein that doesn't belong in the brain crossing the blood-brain barrier, that can easily explain in many of these individuals some of these persistent psychiatric conditions.

And even going back to the adrenal gland, the natural opioid your body makes, the metankeflins, that's one of your happy hormones and mood-affecting as well. So it's multifactorial and multi organs and so unfortunately we do see deposition of spike protein in the brain tissues and this is highly concerning because now you have to consider what medications can we get in there to counter those effects and and neural tissue, yes, it can regrow. There are certain medications that can regrow synapses and whatnot, and what not ketamine, interestingly, is one of those.

But it's difficult to say what are the long-term outcomes in these individuals, because not everything is going to get to the tissues you're trying to treat, especially in these neuroprotected sites. Now, not only are we seeing it in the brain, but we're seeing it in peripheral nerves. We're seeing it in peripheral nerves as well. I have a couple of questions. I'm not clear where the spike protein is getting deposited.

Is it in the myelin, or is it in the cell wall of the neurons, which is such an odd place? I can't think of any other illness that's getting into the neuronal cell wall, per se, without destroying the cell, at least. Yeah, well, and that's the thing. So on the right-hand side of this image, those larger cells, those are the neurons themselves. In the periphery, we do see both myelin destruction, and in some of the patterns, on the one on the photo on the left you will see in in the my well in the white matter as well but yeah so the spike protein is is distributing

everywhere because the brain is so vascular that all the micro capillaries are you know you have to get oxygen to all these neuronal cells. And so the microcapillaries is where it's leaking through into the surrounding white matter as well as into the neurons. So the short answer is yes and yes. It's getting into, and it goes back to the point that that charged lipid nanoparticle loves to hone to fatty tissues. And the brain, again, if someone calls you a fathead, it's a compliment. But I would expect that to still go to myelin. Myelin is a little fattier than the cell wall.

I mean, it's really just very lipophilic. But it's not going to the myelin. It's going to the lipid bilayer. It's weird. It's going to both. It's going to both. And that's why we're seeing a lot of patients with with neuropathies uh post injection and long cove that spike protein homes there again it

causes an autoimmune attack against those tissues and what i think is interesting is that we've been focusing on things like these neuropathies new onset seizures seizures, different neurologic sequelae. But my question is really, what number are we not attributing? What number of neuropsychiatric changes, new onset depression, new onset psychosis, those sorts of things that might actually, when you look at this slide, be related to these vaccines and to the deposition of spike protein? Everyone's happy to say that all of the substance abuse and depression and anxiety is related to the lockdown, and there's no question the lockdown's contributed to that. But what component of this might be actually attributable to to vaccine injury in in the 1920s the the doctor dr menninger himself

felt that everything he was seeing the vast majority of what he was seeing in the early 1920s was from the influenza from 1918 and his the spectrum of psychiatric disorder was vast well and it's it's amazing you can't find what you don't look for. I say that many a time. And if we're not looking, we'll never find it. And so to honor those who have unfortunately passed, be it post-COVID, be it post-vaccine injury, we should be studying those tissues. This should be a primary push medical establishment based on what we've experienced for three years. This is, I'm not the only guy in the world with curiosity, but I have a handful of pathology colleagues around the world that are looking into these mechanisms, and I quote them here because they're esteemed colleagues.

I want to see the funding go into getting the answers because Kelly brings up a great question and the denialism of, oh gosh, there's no such thing as vaccine injury. Well, gosh, I'm showing it right here. And people don't believe me, but the cells, they're screaming at me and they're looking at us in the face and saying, well, here's the spike protein, here's inflammation. And then it goes to Kelly's clinical question of now we're seeing certain manifestations. And I love that you bring up that historic anecdote about the observations after other large pandemics, because how much of, you know, take for example, neurosyphilis. Think of the behavioral changes we see when a pathogen affects

the brain and neurosyphilis, or neuro Lyme disease, neuroborreliosis, et cetera. There's so many pathogens that can affect neural tissue. And we often just describe it to one thing or to nothing. But gosh, we sure have a smoking gun right now. And it would behoove the medical establishment to bring this into consideration when they're making that differential diagnosis on a patient. The other big group of bucket of diagnoses or adverse events, Ryan, I would say are those that fall under the autoimmune ones. And that in my mind includes the increased risk of cancer, given the role the immune system plays there.

We know from the most recent studies, and it's not just one or two, that people who have been multiply vaccinated actually appear to have a significantly higher risk of contracting COVID. We know that there's some clear immune suppression to those people who have been multiply vaccinated. Let's talk about that group of illnesses or diseases that would fall under autoimmune related things and where you're seeing that? Yeah, so first let's jump to the patients getting infected

over and over again. So I'll have Caleb jump to slide number 21 real quick. Everybody hears about antibodies and really a good immune response isn't just antibodies. A good immune response is a good T cell response. So we hear about, oh gosh, I have IgG, I recovered from COVID, or gosh, I have immunity from my vaccine, I formed IgG. Well, you form a lot of different types of antibody and genes get turned on and off and on and off to make certain types of antibodies. Your acute one is IgM. There's an

IgD that stays stuck to the B cells, the plasma cells that make your antibodies. There acute one is IgM. There's an IgD that stays stuck to the B cells, the plasma cells that make your antibodies. There's one in your mucosa, your secretory IgA, which the shots really don't do a good job. If you're trying to prevent a respiratory infection, you want a lot of secretory IgA, and the shots are being put in the body, not into the mucosa. So you don't end up making a lot of secretory IgA. But the one of concern here is IgG4. So in your IgGs, you have one, two, three, four. One and three are your inflammatory ones that will induce like binding to a pathogen and making your body gobble it up. So it basically makes the infected cell or the virus itself yummy. It makes it yummy so that your body will come in and clear it out. Now, IgG4 and IgG2 are actually negative inflammatory. IgG4 we think of in

allergies. And so if I want you to be immune to say a peanut allergen or whatever you're allergic to, you know, a kiddo is allergic to cats or dogs. I had allergy shots as a kid. I was allergic to the world. And you give a little bit of protein over time, a little bit, a little bit, a little bit, and then your IgG4 against that protein goes up and it blunts the immune response. So your immune system says, you know, I'm just not going to hyperreact to that anymore. That's wonderful if it's an allergy. It's horrible if it's a pathogen. Now, there was a study by Dr. Arang in science in December of 22 that showed, unfortunately, after two and even worse after three Pfizer shots, this amount of IgG4 goes up. Well, what does that mean? Now you tolerate the protein, the spike protein. So in this category, now instead of being able to recognize it and fight it off,

your body says, yeah, I'm just going to ignore it. And then Dr. Burra in Germany looked at the Moderna shots and after two shots found the same thing. Now, what was interesting is in the first study, 210 days after two shots, the IgG4 was still elevated. After the third shot, 180 days later, it was still elevated. So what we're seeing is a tolerance to a pathogen. That makes one potentially a sitting duck for other coronaviruses in the future. And if we go to the next slide, please, Caleb, it's not as though we can't see this clinically. This is from the Cleveland Clinic. The more shots you get, the more COVID you get, period. And that's because of immune the more COVID you get, period. And that's because of immune suppression. So you see that top line, those are patients that got three

or more shots. The next line down was three shots. The next line down was, I think two, I can't quite see it. But anyway, you can see the pattern. The upper lines are the people with more shots. And those are people that got more COVID. Now, this was a 51,000-patient study. That's not a small study. And so, you can see this. So, anywhere from two to four times more risk of getting COVID after having these injections. And the more injections you get, the more immune-suppressed you are. IgG4 is one of the reasons for that mechanism. Now, the other thing, you know, you brought up, Kelly, and we can drop the slides now. The other thing you brought up, importantly, is cancer mechanisms. And there's so many here that, you know, it would be an entire show just alone on the mechanisms, unfortunately, there.

But what we're seeing is some immune suppression, and it's persistent immune suppression, especially the more shots you get, the more we're seeing in the laboratory findings, lower T cell counts, lower CD4 counts, lower killer T cell counts. Now this IgG4, yes, it's related to cancer as well. Now this was spike specific in this study, so I'm not going to make any grand leaps or assumptions there per se, but it didn't look at IgG4 across the board in other B cell types. So that's another thing that should be studied, absolutely, because we know in cancer patients, IgG4 is increased and then the ability of the body to say, gosh, you know, I'm going to fight off this cancer. The cells say, no, we're just going to recognize it as self and we're not going to attack it. So it is unfortunate.

And then the Bayer's data bear out that there's absolutely an increase of cancer reporting compared to all other vaccines combined after the rollout of these. And the T cell repair mechanisms are inhibited. DNA repair mechanisms are inhibited. The mitochondria are destroyed and decreased. Hypoxia is one of the mechanisms of cancer growth, et cetera. So there's so many pathways, unfortunately. Everywhere I go in the world, I mean, the amount of reporting I get from oncologists,

radiologists, radiation oncologists, you name it, the cohorts, the young patients that they've never seen cancer in before. Again, if we could open up our national databases, if we had access, it'd be a simple study. Here's the age cohort, every decile. And you look at the type of cancer. I code every day when I make a diagnosis and ICD-10 goes in and says,

here's the cancer, here's the age. And these databases exist. These should be wide open to the public because what has changed in the recent couple of years? Obviously the elephant in the room. And to be clear, as you said, this is not- Kelly, just one quick second coming off the spaces. If you guys, if people are wanting to see the slides

that we're talking about, I know people that are on Twitter spaces realize you can watch it on my Twitter feed at Dr. Drew, also at my YouTube channel, also DrDrew.TV. We're streaming live now, and you can watch the program later if you wish. Sorry, Kelly, go ahead. Yes, and if they're listening to this on the podcast, they can actually get all of this stuff if they just go to DrDrew.com slash 2-1-2023. So it's basically today's date, DrDrew.com slash today-1-2023. So it's basically today's date, drdrew.com slash today's date.

You get the slides. Kelly, sorry. Thanks. What I was going to say, Ryan, just to amplify what you're saying about these cancers, not only are we seeing new onset cancers, very aggressive cancers in far younger age groups, so aggressive invasive colorectal cancers in people in their 30s, invasive melanomas in people in their teens and 20s, lymphomas, leukemias,

myelomas, very unusual cancers in young people. But we also are seeing resurgence of cancers that had long deemed to be in remission. People who had had a breast cancer, for example, you know, go ahead. Yeah, that's a great point. Before you answer, before you answer, are we seeing it? I can't get the data. I mean, when we've always seen cancer as the blossom all of a sudden, I can't get the sudden death data.

I can't get the blossom death data. I can't get the blossom and cancer data. Why is this so obscure? It's the simplest question in the world and to ask it is somehow problematic. That's a great point, Dr. Drew. I mean, why can't we just simply have transparency and data? I have nothing to hide here. I don't have all the answers. I'm asking a lot of questions. I'm certainly seeing patterns. I'm asking my colleagues within the profession, gosh, the techniques I'm doing, these aren't proprietary to me. These are widely available. Why aren't my colleagues doing this? Please do it. Please do it around the world. I have other

colleagues doing it. I'm imploring for transparency and data. So weird. This is humanity at stake. This is humanity. This is why we went into medicine, to help people, not to harm people. So if there's a signal, gosh, let's make that signal transparent, make it manifest. Let's answer the honest question. Whether we like the answer or not, let's ask the question, but where are the agencies? Why are they covering something up? I'm not saying they are or aren't, but it sure seems, like you said, these are not just our database, but other countries have similar databases in terms of health tracking. And this is the job of our CDC, morbidity and mortality tracking. They're supposed to be doing this. This is what we

pay them to do. And I want to jump- We certainly had Dr. Teresa Long on with the defense military database. And she's clearly seeing, as I said, both increase in those cancers in young people and resurgence. Go ahead. Sorry. Yeah. And I get criticized for the, you know, early on, I was the first one in the world to point it out when I started seeing the pattern. And, you know, I said it was X percent. It wasn't quite that. I said the 20-fold. And I went back and looked at my database.

It was only a five-fold, but still it was a five-fold increase in certain types of cancer. I'm like, whoa. So, you know, I self-corrected on that one. If you jump to slide number 13 real quick, Caleb, again, here's, again, a question that needs to be answered. And this is going to your question of myelomas and different lymphoid type cancer. So this is gastric tissue, Dr. Burkhart's slide again out of Germany. And if you go to the next one now, that's all that blue, those are cancer cells. What's inside of every cancer cell? Spike protein.

So again, this begs the question, yeah, yeah, exactly. What? I know. Every B cell there that's malignant has spike protein in it. And so this begs the question, why aren't my colleagues doing these same stains on unexpected cancers in young individual, any individual that has an unexpected cancer. And Kelly brought up a great point. As I travel and lecture and teach, countless doctors say, gosh, I had a patient five years, 10 years in remission. Their cancer is stage four now. They were clear. And all of a sudden, their cancer is back. And that goes,

there's immune mechanisms keeping cancers in check for a lifetime. And all of us have a few atypical cells every day and our immune system just goes and clears them out. That's the job of surveillance of our natural killer cells to poke a hole in atypical cells, throw a hand grenade in, blow them up, get rid of them. But what we're finding is these unusual cancers and unusual age groups and aggressive ones.

And there are mechanisms to explain why, but we should still go back to dr. Drew's question I agree with him a thousand percent open up the data just share the data tell the truth so we can do something for the patient and so we can figure out all these mechanisms so we can prevent harm first do no harm primo no no cherry the oath we all took or help people make it the choice, the informed choice.

That's the part that's eating me up. I can't, informed consent is off the table if we don't have the data. Right, and these are simple laboratory findings. This is giving informed consent. Here's some risks. Here's some obviously post-injection and post-COVID. I'm not going to tease it out into one or the other, but I'm seeing a lot post-injection only as well.

But at the same time, informed consent. And how do you give informed consent if you don't explain all of these potential harms to the patient? Then it's not informed consent. Right. Yeah. And I think that's kind of the profession that I'm so proud to be a part of, to just really beg our colleagues to go back to that curiosity in addition to the caring, or bring back both caring and curiosity. We are sort of running out of time here. Kelly, is there any last minute wrap or questions? No, only that I so appreciate you being here. It's why we have this platform, because we are trying to return not only robust debate, but intellectual curiosity and the kinds of discussions that before this COVID debacle were really the cornerstone of medicine. I can tell you in all of my years, I've been a practicing physician more

than 30 years, I called hundreds and hundreds of times on my colleagues for second opinions, for advice, for their two cents. I never once called the CDC. I never once called the FDA. Or the FDA. I never once called the FDA. No, never. Never called the FDA, never called the NIH. Never, not once. So all of a sudden, we have agencies that are not only just opining, but they are directing this show. And that's not right. I rely on my colleagues. i rely on my own training i rely on the science i don't need federal agencies telling me how to do my job amen

spectacular point yeah and consensus is never the way science is done and that's why we bring in those second and third opinions because a critical thinking doctor that may be seeing something that none of us is thinking about may have that answer for that patient. And so this is why it's so important to ask these questions and not live in fear, but live with a courageous curiosity. Ask the question. I always reserve the right to be wrong. Hey, you have better data. Please bring it so I can learn. And I would hope that our colleagues would act in that same manner. And on behalf of the patient, this isn't about anybody's ego. You know,

some people, MD means minor deity. To me, it means make a difference. So I think that's where we need to get back to making a difference. And we do tend to get, as a profession, we do get into groupthink a little bit. And so the outside the box thinkers are the ones we look to, to break, to shake that up a little bit. Absolutely. And you are. Wrap it up. Yeah. Both of you. Thank you.

Well, thank you for being here. Truly. I know how busy you are and we look forward to having you back for Ryan Cole part three. Hopefully we will, by that point, have come up with maybe some fixes, some ways to actually offer some relief to people who have been harmed both by the vaccines and who are still suffering from long effects of the virus. So blessings. Thank you for being here. Thank you, Dr. Cole. And Kelly, I know you have to run possibly too, so thank you as well. And we will be back with Dr. Kelly Victory next Wednesday with Senator Ron Johnson, so that should be interesting. And apologies, you know, what you're getting us jumping on each other here is there are three different delays going on. And we're trying to time each other so we don't step on each other.

And what happens when you step, you don't hear the other person. You stop what you're saying. And you saw the little Three Stooges thing we got into for a minute there. But that's in the nature of the beast here. So we appreciate you bearing with it when we have little delay issues. Jeff Deist in here from the Mises Institute, which we're going to talk a little Keynesian, or I think he's going to talk a little anti-Keynesian kind of thinking. Michael Schellenberger in on Monday and Wednesday, Senator Ron Johnson. Oh, that's Tuesday. Tuesday, I beg

your pardon. Tuesday, February 7th. Dave Rubin coming in on the 13th. Jessica Rose, Dr. Kelly got her to come in on the 15th. And then Brooke Jackson on the 22nd. Lots of interesting ideas. The process continues to unfold as more and more sort of insight slowly comes to bear. I'm still swimming in a bit of confusion about a few topics. And I can just imagine how tough it is for the average person to know what decisions to make and how to make them. And, you know, it's, it is, it is challenging.

I actually understood what you guys were talking about today. Wow. You're coming along nicely. No, I mean, you, last time I was kind of, it was a little over my head, but this time I was, I was very well. You were kind of ready for it. I think the you were kind of ready first i think the first episode if you haven't seen the first episode you should see it but we had a million views it really was well received but um it's starting to make sense to me good a little bit okay good but i do intermittent fasting okay good and i i'm so glad that that's what helps get the,

and taking care of your health, obviously. But, um, that was interesting. It is interesting. Well, we appreciate you all being here. Those of you on Twitter spaces, of course, those of you on the restream and Rumble Rants, we were watching you. And in fact, Susan, you didn't see this, but I, uh, was so engaged over at the restream. I heard. I didn't come back. I was at my head of my computer. You can go back and watch. They're probably worried you had a stroke or something. We were all trying to get his attention, but he didn't have his headphones in. So we couldn't get his attention anyway. To me, it sounded like the guy, you know, one of those,

the guy that was just been talking a few seconds earlier on the ad. So, Caleb, anything from your perspective? No, I just, I always love when Dr. Cole comes on because he explains these things so well and so patiently that it's, I can be running all of these controls and I can still learn something and understand what he's saying. I really appreciate it when he comes on the show. Right, right. It doesn't go over your head the nerdier the better though that's what i i know that's my motto we can we can we can go further if you want to well i think people are really listening like you know people come on the restream and you know people come and go but i think people really want to hear the message and they're getting, you know, educated. It's sort of like taking a class, you know, you really learn something. Yeah, good. And again,

my position remains, people are always astonished, but it remains that I still vaccinate my elderly patients. I'm convinced they have had a significant benefit from it. I've just been walking, getting a couple of people in very complex medical situations through COVID, and the fact that they were fully vaxxed helped the situation, trust me, because I was unable to use Pax Levita or Molnupiravir because of the complexity of the situation medically. And so I haven't seen any side effects in elderly patients. I've seen lots of stuff in younger patients, which is why I've been concerned about it and been wanting to talk to all these people that have ideas about it to see if we can figure out what is going on.

But my greatest source of frustration is something Dr. Kuhlman and I just talked about a few moments ago, which is we don't have, the data does not, it's, it's, it's, the data does not appear transparent. It's too soon. There appears to be something, just ask a simple question. We need a couple of simple questions asked, and no one has done. The only way, as he said, if you don't look, you can't find. And if you're not asking a question, you don't know what you're looking at.

You have to come at it with a question, a question like, are there increases in sudden death in 35-year-olds? Are there increase in sudden recurrences at stage four of remission in cancers in 75-year-olds? Is this happening? Yes or no? And I've not seen anybody asking questions like that. It is deeply, deeply concerning to me. So in the meantime, we'll keep fishing around, trying to figure out exactly what's going on here. And as Kelly said, with the one database we do have is the one through the military because it's so carefully kept.

Teresa Long does see some of this there, so that is what you call a signal. We need a bigger study on that. All right, everybody, we'll see you tomorrow with Mr. Deist. I think he pronounced his name. 3 o'clock Pacific time. Until then, mahalo.

Ta-ta. Ask Dr. Drew is produced by Caleb Nation and Susan Pinsky. As a reminder, the discussions here are not a substitute for medical care, diagnosis, or treatment. This show is intended for educational and informational purposes only. I am a licensed physician, but I am not a replacement for your personal doctor, and I am not practicing medicine here. Always remember that our understanding of medicine and science is constantly evolving. Though my opinion is based on the information that is available to me today, some of the contents of this show could be outdated in the future. Be sure to check with trusted resources in case any of the information has been updated

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