Bite Back with Abbey Sharp - Can GLP-1s Treat Eating Disorders? One Woman’s Controversial Story Is Challenging Everything

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Starting point is 00:00:23 If you were someone you know is concerns about gambling, visit connectsontario.ca or call 1866-5-3-1-2-600. Terms and conditions apply. How do you know that the medication treated your eating disorder rather than simply treating one symptom by suppressing your appetite so effectively that you couldn't binge and purge? Welcome to another episode, A Bite Back with Abby Sharp, where I dismantled Diet Culture Rules, call it the charlatans spinning the pseudoscience, and help you achieve food freedom for good. For decades, people who binge ate were told some version of, you just need more willful. power. Like, just stop being such an undisciplined glutton and you'll lose weight. Thankfully, we now know it's much more complicated than that. We know that restriction,

Starting point is 00:01:14 trauma, dieting, emotion, regulation, genetics, neurobiology, and our food environment can all play a role in binge eating. But as conversations around GLP-1s and food noise have gone mainstream, I think there's another uncomfortable question we now need to In moving away from the willpower explanation, have we sometimes become too confident about what is driving the behavior? Because we talk a lot about food obsession and binging as consequences of restriction, dieting, trauma, and unmet emotional needs. And for many people, they absolutely can be. But what about the person who stops dieting, adequately nourishes themselves, challenges their food rules, understands their triggers, goes to therapy and does all the psychological recovery work, and the volume on food is still screaming.

Starting point is 00:02:10 What if, for some people, there's an additional biological component to that drive that we haven't adequately understood or treated? Today's guest Paige Gordon sits on the board at Nita after living with bulimia for 22 years. After starting a low-dose GLP1 medication, she says something, extraordinary happened. The drive to binge and purge disappeared. Now, to be extremely clear here, that does not mean that GLP-1s are a proven treatment for bulimia. They are not. The research is nowhere close to that. But her story raises a question that I don't think we should be afraid to ask. Have we sometimes been treating a biologically amplified drive as though it were primarily a thought

Starting point is 00:02:59 pattern that someone just needed to unlearn? And on the flip side, are we now at risk of swinging too far in the other direction and turning a deeply complex psychiatric illness into a hormone or reward circuitry problem that we can simply medicate our way out of? Because that is the uncomfortable gray space I want to live in today. All right, and before we start, I just want to take a moment to thank you all so much for joining me today. If you are new here, Welcome to the Byteback family. Please do take a moment to leave me a little five-star review and a little love note and subscribe on Apple, Spotify, YouTube, or wherever you are listening and watching so that you never miss out

Starting point is 00:03:44 on an episode. All right, kids, let's do this. Hage, thank you so much for joining me today. I'm really excited about this conversation. I'm glad to be here. Thanks for having me. Okay, so take me back before the GLP1. What did the urge to, you know, binge or purge actually feel like in your body and brain?

Starting point is 00:04:05 So before I started purging, which started when I was around 18, I would binge. And I started binging when I was 11. And so I was a binger for a good seven years before I started purging. And it had really nothing to do with feeling hungry. That would be the first thing I would say. It was an impulsive, oh, my. almost like something that was on my mind all the time in terms of a certain type of food. I tended to go for the highly ultra processed food or highly satiable food that were high in sugar

Starting point is 00:04:41 and fat. You know, I certainly wasn't binging on broccoli. And it just kind of became this obsession in my mind where if I didn't give in to those behaviors, then it was difficult for me even to function. And it had very little to do with being hungry. I could eat three meals a day and I still found myself at the donut shop eating two dozen donuts, for example. And so in that way, it was almost maybe a little bit like OCD.

Starting point is 00:05:08 I mean, we called this food noise now, but that's not a new concept in the eating disorder field for sure. They just used a different name. And I had that. Yeah. Yeah. Yeah. And just for folks who, you know, hear this term now, food noise over and over again, like,

Starting point is 00:05:25 we're not talking about like, oh, what's for dinner or, you know, I can use a snack. We're talking about relentless mental chatter about food that can crowd out cognitive space for creativity and relationships and work and self-care and a million other things like you just said. Like you can't focus on anything else until you kind of give into that, what that food noise is asking for. So in your experience, was there a point where you, you know, like you generally, genuinely were not restricting anymore. You were adequately nourishing yourself, like you said, you're doing the recovery work and the food obsession was still there. Always. Yeah. So for me, so I had, I'm 44 years old. So I had been eating disorder in the 90s. That wasn't in the DSM yet. So I don't think anyone would have known what to

Starting point is 00:06:15 do with me if I had asked for help. And when I first went into treatment, which was in 2000, the treatment was definitely based on a restrictive modality, which is, you know, good evidence. I don't think I'm the normal in terms of what's typical. So I'm not, I'm not criticizing treatment in that way. I just was an outlier, I suppose. And so, yeah, when I first started treatment, I was in a larger body. I was not restricting. You know, the first time I learned about sizes was actually in like quantities of food was

Starting point is 00:06:49 in treatment. Like before treatment, I had no idea that a half of banana was one serving size of fruit. I would eat a normal, I would eat a banana like a normal person. And ever since treatment in 2000, now I'm like, oh, half a banana, that's a certain. You know, this information I didn't need. But I learned it an eating disorder treatment, which is funny. So, yeah, I did never restrict. And that was always the basis of what the, I kept being told if I just ate three meals, two snacks a day, that this urge, this craving to binge would go away.

Starting point is 00:07:19 and that wasn't my reality. And so I think probably that was the miss. No one ever asked me in treatment if I binged before I started purging ever. And I don't know if that happened because I went into treatment early in the 2000s. I'm not sure if it would be different now. But it was always assumed that, you know, my bulimia was the way it was because of I was restricting and it just wasn't true. Right.

Starting point is 00:07:42 Yeah. And I think one thing that the GLP one era has really forced us to confront here is that, you know, we may be lumping very different experiences together under this umbrella of food noise, because, you know, as you're kind of alluding to, we've long known that restriction itself can make the brain extremely preoccupied with food. But now we're learning more about, you know, genetics and biology and how that can influence hunger and satiety and reward sensitivity and susceptibility to weight gain. So I'm so glad we're now having that conversation. But let's talk about the GLP1 now. So what changed for you, symptomatically, psychologically, when you started the

Starting point is 00:08:25 GLP1? Sure. So I take a very low dose, first of all. I take sub-therapeutic of what you would, what's FDA approved for weight loss, more than half less than the starting dose, for example. But I would say around week three, just that urge to binge completely went away. It was completely off my mind. And because I didn't have this overall. writing biological signal that was like driving me towards this behavior. I was for the first time in my adult life able to make better choices for myself because I always knew what I needed to do. I knew I, you know, I make a joke, Abby, that like I have probably more hours than dietitians than some dietitians do at this point because I was in and out of their treatment for

Starting point is 00:09:10 which I think is pretty common for people with eating disorders. They're pretty well educated on nutrition. You know, so I knew what I needed to do. I just couldn't get myself to do. it. And so once I kind of, the GOP helped turn the volume down. And so I felt like I had more agency to make those better choices. I was really motivated not to have my eating disorder anymore. So as soon as that dial went down just a little bit, I was very easily able to be like, okay, three meals, two snacks, three meals, two snacks. And I did that right away. And even if I didn't necessarily want to, but similar to what you do when you're first going to treatment, right? I just did it myself. And I was able to stick with that.

Starting point is 00:09:49 You know, in my case, I actually gained a little weight because I was, you know, and I was fine with that. I just didn't want the urges to binge. And for the first time, it actually worked. And I was like, oh, my God, I think we might be missing something for a subset of patients, certainly not most. And I don't know how many. But I felt like it was a scientific question worth investigating. Oh, yeah. I think this is where your story is so interesting and complicated in some ways,

Starting point is 00:10:19 because it raises this possibility that the exact same surface complaint, which is, you know, I can't stop thinking about food, could arise from very different mixtures of deprivation, reward sensitivity, aptite signaling, learned behavior, compulsivity, psychological distress, like so many different factors here. And theoretically, the appropriate intervention could be completely different depending on what's driving it. So, you know, for one person, those intrusive thoughts might be their, brain appropriately screaming like, feed me, you know, because they are depriving themselves. So suppressing that signal further via a GLP1 could be extremely dangerous. Absolutely. But then for another

Starting point is 00:11:00 person, like perhaps in your situation, reducing that intensity, that food salience of the intensity of that food salience and the reward-driven urges might be super therapeutic. And unfortunately, you know, as we know right now, we don't have a validated clinical tool that can confidently tease those experiences apart. And just looking at someone or weighing them is not evidence of what is going on in their head. It's just not. And so I love your story because I think it complicates the status quo because I imagine a lot of eating disorder clinicians would ask, you know, how do you know that the medication treated your

Starting point is 00:11:44 eating disorder rather than simply treating one symptom by suppressing your appetite so effectively that you couldn't binge and purge. I'm curious, what do you think of that suggestion? Did you initially interpret your experience as proof that your illness had been biological all along? So I think there was a couple questions there. The first one in terms of clinical responses or clinician's responses to me, I think it's a valid question. And I think that that can happen in some instances and where you're just completely eradicating your hunger cues. And so then you're just sort of on autopilot. Even in that instance, though, I wonder if there's not an opportunity for that to be

Starting point is 00:12:29 a helpful intervention in some instances, as long as someone is being properly monitored to stop the behavior and to get them on the new habit, right? So, I mean, that's an if question. I don't think we should be, like, trialing this on people randomly. but I wonder even in that instance if there aren't like some specific instances that could be helpful. Certainly for me, I had been sick for 22 years. So by the end of my disease, most of my meals were binges and then purging. And so I think it did kind of stop that behavior because at that point it had gone from binge eating to, you know, making a bad situation a lot worse with my stomach being stretched out and all the things.

Starting point is 00:13:09 So it really was able to stop that and make new habits for myself. But, you know, like the gastric emptying portion of taking a GLP, that goes away in about a month or so. It's a short-term effect that is known. So the rest of that is all psychological. So for me, I saw it as evidence that there was something biological going on with me. However, I would argue that all eating disorders are biological. And what I mean by that is this conversation we're having right now is a neurological event. So I don't think it's like, oh, it's based in biology or genetics or, oh, it's based in an environment and social cues.

Starting point is 00:13:49 They're all together. I mean, whether or not you're having preoccupation with food or if you have body dysmorphia or whatever, that is still something going on in your brain. So it's all biological. And I don't think those two things are like at odds with each other. But for me, certainly, this is kind of crazy. Abby, but like you would think that I would know that I was mentally ill because I had an eating disorder for 22 years. And I did.

Starting point is 00:14:15 But I was like, you know, I knew what I was doing. In some ways I wish I would have had, you know, that I didn't realize what I was doing. It would have been easier because it just made me hate myself more because I couldn't stop the behaviors. But taking the GLP medication was such a profound effect in terms of even just affecting the way my mood was. It was the first time I was able to. to confidently admit that I had a mental illness

Starting point is 00:14:41 because I had a comparison point to how I felt in my brain and my body before versus after. And I was like, wow. I'm like, it did for me what they were hoping 60 milligrams of Prozac would do, right? Right. That didn't work at all. I tried that.

Starting point is 00:14:57 It did absolutely nothing for me. And so I was like, wow. There is something so profoundly dysregulated in my brain that this medication is somehow indirectly helping It's probably not fixing the exact thing, but it's helping just enough for, and I'm really motivated, so I was able to make change. Like, it was just amazing. It was amazing.

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Starting point is 00:16:45 And so tell me more about that, the kind of like holistic effects, the holistic success of a GLP1. What happened to body image and food rules or need to compensate and purging and weight obsession? Like what about all of those elements that we know are associated with eating disorders? Right. So I've had some people tell me they didn't think I had bulimia because I didn't have obsession with body image. and that's one of the requirements for having bulimia in the DSM. Some people tell me that they think I had food addiction, which I'm like, eh, I was throwing up like three or four times a day.

Starting point is 00:17:21 Are you sure I didn't have bulimia? You know, so it's one of those things. I think that's probably one of the problems at DSM is like all based off of symptoms, you know? And, you know, anyway. So it's a tricky question. Fat phobia and weight bias certainly contributed to me getting bulimia. There's no question. I started gaining weight because of my binge eating disorder.

Starting point is 00:17:46 And what I sincerely thought to myself is like, I'm going to end up being one of those people that is so disabled, they're not going to be able to physically walk. And they're going to have to get craned out of a house. I was like, that's what's going to happen to me. I can't control myself. So if that's the way I'm going to be, I might as well not be that and be thinner. I wasn't necessarily going like, oh, I need to be a size six. I need to be size eight.

Starting point is 00:18:11 I just didn't want to be a thousand pounds. So that contributed to my original decision to start purging, unfortunately. So I had weight obsession in that way. Like I was afraid of being obese. And there's no question about that. And, you know, that's something I've had to really think through of like, why did I feel that way? Like what messages were told to me as a young child that made that seem like worse than having bulimia? That's quite sad.

Starting point is 00:18:38 Yeah. But I never had I wasn't I never weighed myself multiple times a day. I wasn't obsessed on anything like that. So that didn't change because I didn't have that. I've never counted calories. Thank God because that's something you can't unlearn. So I'm lucky in that way. So I, you know, that didn't change.

Starting point is 00:18:54 What did change, I would say, is there is a long time where I would avoid certain food, not because I was afraid of weight gain because I knew it just putting it in my mouth would be an instant binge similar to like an alcoholic. not wanting the first drink. So it wasn't related to me being afraid it was going to make me fat, but I just knew it would turn into a binge. And so once I took the GLP, it really opened up what I was able to eat and enjoy, sincerely enjoy, because now I can eat absolutely anything,

Starting point is 00:19:25 and I don't have to worry about it turning into a binge. So it really opened, I eat way more food than I ever did before. It's kind of amazing. That's amazing. Yeah, it's so interesting because, like, again, we don't have validated tools for this yet, But I feel like when it comes to that earlier question of like, could a GLP1 actually treat an ED as opposed to suppressing a symptom? I think, you know, perhaps one litmus test or question to ask yourself could be something like, you know, if this medication had completely eliminated all binging and purging and giving you your life back, but you hadn't lost a single pound, would you consider equally successful? Because I think if that the answer is yes, then I think that is a really incredibly important distinction.

Starting point is 00:20:09 Because now we're not talking only about a weight loss treatment that happens to reduce binging. We're talking about whether there could be theoretically some kind of, you know, some psychiatric or behavioral therapeutic effect independent of weight loss. And that's really powerful here. That is what I want to see, you know, invest. I'll take it a step further. So I, you know, I think I just had a Dexa scan and my body max indexes. It's within the normal range, but it's on the lower end. If someone, because this is so confusing to people, this concept, if I went to eating disorder

Starting point is 00:20:47 clinic and they told me that the only way they would give me this medication is if I was no longer weight suppressed, because I am weight suppressed, meaning when I first went into treatment, I was like 180 pounds. I'm now around 125. I would think you were stupid for telling me that because I think it would add a whole other host of side effects that aren't positive. But I would do it. I would do it in two seconds.

Starting point is 00:21:09 I would be in a larger body, no problem if it meant that I didn't have to have this urge and this craving all the time. And that's what I always had a problem with in treatment. I was like, I don't have a problem being in a larger body, but it's not going to solve my problem. I'm just going to be in a larger body and I'm still going to have this obsession with food. So like, why are you guys making me do this? So, yeah, I would do that in two seconds if it's what I needed to do.

Starting point is 00:21:33 That's amazing. I think that's very telling, right? Now, you now sit on the border at NIDA, and I assume you're sitting beside a lot of eating disorder clinicians who understandably are reluctant to entertain this idea of giving GLP1 medications to people with eating disorders. And I think there are real concerns here about restrictive eating and nutritional inadequacy. and weight loss reinforcement and fat phobia and the normalization of ultra-thinness that has come with this JLP1 boom that we're seeing. Yeah.

Starting point is 00:22:07 Now, what are the questions that you think the eating disorder community, like, is afraid to ask that may stand in the way of us fully understanding, you know, binge biology and improving treatment? Well, it's hard, it's hard to take a look in the mirror and be realistic of where we are in treatment. I would say that's the first thing. Anytime I make a post, like if a paper comes out, like there's been two papers recently. One came out recently about like why a treatment hasn't

Starting point is 00:22:38 improved in 20 years and it like listed out all the reasons. And then another one came out in December. I met an analysis that said like, you know, in terms of binge eating, the outcomes have actually gotten worse in treatment unless. So anytime I do a post on that, like the clinicians start coming for me, you know, because I think it's really hard. to have a realistic look in the mirror and say, like, what we're doing isn't working that well. It's almost treatment resistance is the norm. And so if that's the case, then it means we're missing something. It's not the patient's fault.

Starting point is 00:23:12 It means we fundamentally don't understand something. And I think that anything that could potentially reduce suffering deserves rigorous clinical investigation. I'm not suggesting that people go try this at home. You know, what I'm suggesting is that this deserves investment from the scientific community to see if there's something there. And wouldn't it be great if we could find a way to figure out ahead of time if someone is going to respond well to this medication instead of causing harm? Because right now, I have no idea if it's causing more harm than good. You know, I've had 85 women contact me say they love it, but that doesn't mean anything. That's a biased response.

Starting point is 00:23:55 And then I have other people say clinicians are like, I'm talking to people with anorexia. And it's causing someone. I'm like, well, that doesn't mean anything either because most people would go into treatment have anorexia. You're not seeing people with Benjiating disorder. Yeah. So we don't have a good sense of it. And I would like to know.

Starting point is 00:24:10 I'd like to know. And for you, are you worried that your story could be weaponized by the weight loss industry? I mean, like I can just imagine some telehealth company seeing a story like yours and turning like this medication changed my relationship with binging and purging into gelp1 is now treating food addiction and eating disorders when that's not what the evidence says yet right they want to uh i'm not worried about it at all because pharma wants to stay far far away from this topic right now um you know historically pharma has abandoned eating disorders that's why we only have two approved FDA medications which were both repurposed drugs um you know they don't they don't want to invest in us some reasons are we're

Starting point is 00:24:50 difficult, all mental health are difficult to run clinical trials on. And the group is too small. They don't see it as having any money. So for the most sort, they want to stay away from us. So I'm not particularly worried about that, to be honest. I do worry, and it was a big worry of mine when I wrote my book, that someone would read my book and pick it up as a prescriptive thing. And I was really, really worried about that. I'm super clear in the foreword. And I'm very clear throughout the book that this isn't prescriptive. What it is is a call to action with researchers and clinicians to ask better questions. And that's pretty much it. Although there are doctors who prescribe this off-label and, you know, off-label medication is not

Starting point is 00:25:31 illegal. You're allowed to do that. But definitely don't try it at home. No, you know, of course. Yeah. I mean, I guess there's also the kind of risk that someone with a restrictive eating disorder could hear your experience and then use it to justify to themselves or to their healthcare care provider, like I should be prescribed a JLP1. And, you know, so that's where it's, again, we need better standards of care for folks with e-dissorders and more research so that, you know, clinicians do have, you know, have an understanding of when they can be used and when they should be used and when they may do more harm than good.

Starting point is 00:26:07 And I think, you know, I think it's also the responsible thing to point out that the opposite risk exists of this conversation too, because JLP ones now have become so dominant in conversations that we may reduce all problematic eating behaviors as just some kind of biological reward circuitry problem where you kind of said earlier, like it's not one or the other, which these eating disorders are the result of a complex interplay of perhaps that circuitry problem, but also dichulture, trauma, learn behavior, food environment. Right. And so I think it's important that we don't replace psychological, you know, reductionism with biological reductionism. For sure. And we, you know, the bio, psychosocial, we shouldn't be picking whichever one agrees with our own kind of personal ideology. We, a good health care provider. Yeah, we need to be investigating all three. And so I guess what I would love to hear from you, what exactly do you want researchers to? investigate? What is the most important questions we need to be asking next?

Starting point is 00:27:17 A great question. So I'm pushing really hard for feasibility studies right now to see if these medications are appropriate to be used in specifically weight suppressed delimary nervosa patients and patients with binge eating disorder. That's sort of like the first step before you would go to a randomized control trial. So I think that's really important. We're having trouble getting investment. I mean, I've personally ponied up money to say that I will throw money towards this problem. So that's the first thing, because we can argue about this all day long, but, you know, we don't have any, if we don't have any money to study it, like, what are we doing? The second thing is, is I think, really more investment in understanding about rare variants. So the Broad Institute at

Starting point is 00:28:01 Harvard is doing a large genetic study right now, and it's free to submit your swabs. They're trying to look into that. I have a rare variant on my GLP pathway that we think is associated with disorder eating. And, you know, in my very simplistic best case scenario, wouldn't it be wonderful if we could identify some of these gLP variants that would be a predictor of response to these medications that we would know ahead of time? Pharma is not going to support that type of study. That would have to be independent because they would rather get FDA approval for a broad group that works okay instead of it working extraordinarily well for a very small group. So it's really going to have to be independent funders that come up and bring the money to the

Starting point is 00:28:48 table to get it done. But I think that's the most interesting questions that I have, about precision medicine, really. Because I think that paper that I alluded to from Dr. Levingston that came out about treatment outcomes not improving in the past 20 years, one of her main points was that we're treating to averages and we're not having precision medicine. And that's It's one of the reasons where we are. Yeah. And when it comes to eating disorders specifically, you know, there is no one-size-fits-all. I know that we have, you know, we have specific criteria to hit in the DSM, but every individual's

Starting point is 00:29:21 experience is so highly variable, what their triggers are, what their origin story is, what specific, whether they're, you know, we're now talking about genes and variants and the intersection of those variants with, you know, their food environment and diet culture and what they're exposed, to you and all of those messages. And so I think, yes, an individualized approach is going to be so critical. And so I'm very, very appreciative of, you know, the work that you're doing, the advocacy that you're putting forward. And also for coming on and sharing your journey. This was such a fascinating conversation. Yeah, this is fun. And can you tell everyone where they can find more information about what you're doing and in any way they can support you? Great. Thank you. So my

Starting point is 00:30:06 website is pagecordon author.com. You can pre-order my memoir that's coming out. So 100% of the royalties to this book are going, donating back to NIDA, specifically to a fund that's supporting research on this topic. So it's really just important to me in that way. And I always like to point out the NIDA website, because they have a wonderful screener tool there. If you think you're having any issues with food. You can go on there and they will give you resources for next step so you can talk to professional. So yeah, my website's the best way. And then I'm also on Instagram. I'm really vocal on there. Same name, Paige Gordon author. And the last thing, you've touched on it, but I just want to make sure I'm really clear that as much promises these medications may have for binge eating

Starting point is 00:30:56 type disorders, it's the absolute opposite for restrictive type eating disorders. And I say restrictive type because people that have been geneating disorder can have restrictive binge eating disorder. So anyone that's coming from a place of restriction, you've got to stay really far away from these medications. And anyone even with, you know, that it could be promising, if you're thinking about using it in a weight loss context, it's also bad news bears. Yeah. It's a red flag.

Starting point is 00:31:26 Yeah. So I just always want to tell that to people. Yeah. I appreciate just kind of nailing that home because absolutely we don't want this. And that is what I was kind of alluding to. We don't want this conversation to turn into, oh, well, you know, this is kind of, you know, my way to get a GLP1, which is obviously not what we want to be endorsing. So, again, thank you for that kind of hammering home that disclaimer. And thank you again for what you do.

Starting point is 00:31:52 Thank you. Thank you so much. It was a lot of fun. Okay, this was such a unique and interesting conversation. And, you know, as someone whose only encounter with food noise was through extreme diet restriction in, you know, my teen years when I had an eating disorder, I am always so eager to learn from those with varying food journeys. And I'm very, very much invested in the future of GLP1 research and the advocacy work that Paige and others are doing to help us make these medications safer and more effective for those who use. them. And while we have loads of research on their use for obesity and metabolic disorders, we are still a very long way from seeing official label expansion for various EDs. And the lack of

Starting point is 00:32:43 research really is evidence of that. So a recent 2026 review found only nine empirical studies looking specifically at GLB1 medications in people with eating disorders or disordered eating. And most of that work has focused on binge eating, so not bulimia or anorexia, orthorexia, arphid, or other combinations of eating disorder presentations. But what we do have for BED or binge eating is definitely quite interesting. So a recent meta-analysis of five small studies involving 182 people did find that these drugs may help to reduce some symptoms in some people with BED. But in addition to being still very small and heterogeneous, they generally are studying weight loss as a primary outcome with eating disorder symptoms as kind of almost side effects. And that leaves us with some pretty massive unanswered questions.

Starting point is 00:33:41 So we still don't know whether GLP-1s are actually modifying eating disorder psychopathology or simply making binging harder by lowering aptite and food reward. We don't know who might benefit versus who might become more restrictive. We don't know what happens to body image distress or compensatory behaviors or nutritional adequacy over years of these medications use. And we have remarkably little evidence in bulimia specifically. The newest reviews are essentially saying the same thing. Promising signal, major safety and misuse concerns, and not enough evidence. yet to guide routine eating disorder treatment. So I don't think that Page's story gives us an answer

Starting point is 00:34:29 yet, but I do think it gives us a really good research question and some hope that future treatments won't put all people with binge type symptoms into an existing explanatory box. We clearly need more studies designed to figure out who is binging, why they're binging, what exactly the medication changes, and whether that change actually looks like recovery. And I will absolutely be keeping my finger on the pulse as more unfolds. But on that note, that's a wrap for today. If you have not already, please make sure to follow or subscribe wherever you get your podcasts. Do leave me a nice little five-star review and a comment.

Starting point is 00:35:09 And until next time, signing off with Science and Sass. I'm Avi Sharp. Thanks for listening. My rec league goalie says he plays for the love of the game. You gave up six goals last week, Steve? Maybe play to win, huh? Like with Bet365, I get paid the second my team goes up big with early payout and can cash out at any time before the game ends.

Starting point is 00:35:35 Come on, Steve, bet 365, the home of winning early. Must be 19 and older, Ontario only. Please play responsibly. If you were someone you know is concerns about gambling, visit connectsontario.ca or call 18665 through 1,2,600. Terms and conditions apply.

Bite Back with Abbey Sharp - Can GLP-1s Treat Eating Disorders? One Woman’s Controversial Story Is Challenging Everything

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